John E Harris

University of Massachusetts Medical School, Worcester, MA, United States 
dermatology, immunology, autoimmunity, T cells, cytokines, vitiligo
"John Harris"
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Chong SZ, Evrard M, Devi S, et al. (2016) CXCR4 identifies transitional bone marrow premonocytes that replenish the mature monocyte pool for peripheral responses. The Journal of Experimental Medicine. 213: 2293-2314
Rork JF, Rashighi M, Harris JE. (2016) Understanding autoimmunity of vitiligo and alopecia areata. Current Opinion in Pediatrics
Li JL, Lim CH, Tay FW, et al. (2016) Neutrophils Self-Regulate Immune Complex-Mediated Cutaneous Inflammation through CXCL2. The Journal of Investigative Dermatology. 136: 416-24
Harris JE. (2016) Cellular stress and innate inflammation in organ-specific autoimmunity: lessons learned from vitiligo. Immunological Reviews. 269: 11-25
Harris JE. (2015) Melanocyte Regeneration in Vitiligo Requires WNT beneath their Wings. The Journal of Investigative Dermatology. 135: 2921-3
Ezzedine K, Sheth V, Rodrigues M, et al. (2015) Vitiligo is not a cosmetic disease. Journal of the American Academy of Dermatology. 73: 883-5
Zhang R, Borges CM, Fan MY, et al. (2015) Requirement for CD28 in Effector Regulatory T Cell Differentiation, CCR6 Induction, and Skin Homing. Journal of Immunology (Baltimore, Md. : 1950)
Agarwal P, Rashighi M, Essien KI, et al. (2015) Simvastatin prevents and reverses depigmentation in a mouse model of vitiligo. The Journal of Investigative Dermatology. 135: 1080-8
Richmond JM, Harris JE. (2014) Immunology and skin in health and disease. Cold Spring Harbor Perspectives in Medicine. 4: a015339
Rashighi M, Agarwal P, Richmond JM, et al. (2014) CXCL10 is critical for the progression and maintenance of depigmentation in a mouse model of vitiligo. Science Translational Medicine. 6: 223ra23
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