Francisco J. Sánchez-Rivera
Affiliations: | 2008-2016 | Biology | MIT / Koch Institute |
| 2016-2021 | Cancer Biology & Genetics | Sloan Kettering Institute, New York, NY, United States | |
| 2022- | Biology | MIT / Koch Institute |
Area:
Cancer BiologyWebsite:
https://biology.mit.edu/profile/francisco-j-sanchez-rivera/Google:
"Francisco Sánchez-Rivera"Parents
Sign in to add mentor| Tyler Jacks | grad student | 2009-2016 | MIT / Koch Institute |
| Scott Lowe | post-doc | 2016-2021 | Sloan Kettering Institute |
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Publications
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| Leibold J, Tsanov KM, Amor C, et al. (2024) Somatic mouse models of gastric cancer reveal genotype-specific features of metastatic disease. Nature Cancer |
| Torborg SR, Grbovic-Huezo O, Singhal A, et al. (2023) Solid tumor growth depends on an intricate equilibrium of malignant cell states. Biorxiv : the Preprint Server For Biology |
| Karagiannis D, Wu W, Li A, et al. (2023) Metabolic reprogramming by histone deacetylase inhibition preferentially targets NRF2-activated tumors. Cell Reports. 43: 113629 |
| Ely ZA, Mathey-Andrews N, Naranjo S, et al. (2023) A prime editor mouse to model a broad spectrum of somatic mutations in vivo. Nature Biotechnology |
| Karagiannis D, Wu W, Li A, et al. (2023) Metabolic Reprogramming by Histone Deacetylase Inhibition Selectively Targets NRF2-activated tumors. Biorxiv : the Preprint Server For Biology |
| Hu J, Sánchez-Rivera FJ, Wang Z, et al. (2023) STING inhibits the reactivation of dormant metastasis in lung adenocarcinoma. Nature |
| Soto-Feliciano YM, Sanchez-Rivera FJ, Perner F, et al. (2022) A molecular switch between mammalian MLL complexes dictates response to Menin-MLL inhibition. Cancer Discovery |
| Li X, Huang CH, Sánchez-Rivera FJ, et al. (2022) A preclinical platform for assessing antitumor effects and systemic toxicities of cancer drug targets. Proceedings of the National Academy of Sciences of the United States of America. 119: e2110557119 |
| Sánchez-Rivera FJ, Diaz BJ, Kastenhuber ER, et al. (2022) Base editing sensor libraries for high-throughput engineering and functional analysis of cancer-associated single nucleotide variants. Nature Biotechnology |
| Romero R, Sánchez-Rivera FJ, Westcott PMK, et al. (2022) Publisher Correction: Keap1 mutation renders lung adenocarcinomas dependent on Slc33a1. Nature Cancer. 1: 935 |