Katrina L. Bogan

Affiliations: 
2010 Biochemistry Dartmouth College, Hanover, NH, United States 
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"Katrina Bogan"

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Charles Brenner grad student 2010 Dartmouth
 (Discovery of new NAD+ metabolic pathways and nutritional regulation.)
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Publications

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Belenky P, Stebbins R, Bogan KL, et al. (2011) Nrt1 and Tna1-independent export of NAD+ precursor vitamins promotes NAD+ homeostasis and allows engineering of vitamin production. Plos One. 6: e19710
Evans C, Bogan KL, Song P, et al. (2010) NAD+ metabolite levels as a function of vitamins and calorie restriction: evidence for different mechanisms of longevity. Bmc Chemical Biology. 10: 2
Bogan KL, Evans C, Belenky P, et al. (2010) Identification of Isn1 and Sdt1 as glucose- and vitamin-regulated nicotinamide mononucleotide and nicotinic acid mononucleotide 5′-nucleotidases responsible for production of nicotinamide riboside and nicotinic acid riboside (Journal of Biological Chemistry (2009), 284 (34861-34869)) Journal of Biological Chemistry. 285: 3524
Bogan KL, Brenner C. (2010) 5'-Nucleotidases and their new roles in NAD+ and phosphate metabolism New Journal of Chemistry. 34: 845-853
Bogan KL, Evans C, Belenky P, et al. (2009) Identification of Isn1 and Sdt1 as glucose- and vitamin-regulated nicotinamide mononucleotide and nicotinic acid mononucleotide [corrected] 5'-nucleotidases responsible for production of nicotinamide riboside and nicotinic acid riboside. The Journal of Biological Chemistry. 284: 34861-9
Bogan KL, Brenner C. (2008) Nicotinic acid, nicotinamide, and nicotinamide riboside: a molecular evaluation of NAD+ precursor vitamins in human nutrition. Annual Review of Nutrition. 28: 115-30
Tempel W, Rabeh WM, Bogan KL, et al. (2007) Nicotinamide riboside kinase structures reveal new pathways to NAD+. Plos Biology. 5: e263
Belenky P, Racette FG, Bogan KL, et al. (2007) Nicotinamide riboside promotes Sir2 silencing and extends lifespan via Nrk and Urh1/Pnp1/Meu1 pathways to NAD+. Cell. 129: 473-84
Belenky P, Bogan KL, Brenner C. (2007) NAD+ metabolism in health and disease. Trends in Biochemical Sciences. 32: 12-9
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