Year |
Citation |
Score |
2021 |
Azad P, Villafuerte FC, Bermudez D, Patel G, Haddad GG. Protective role of estrogen against excessive erythrocytosis in Monge's disease. Experimental & Molecular Medicine. PMID 33473144 DOI: 10.1096/Fasebj.2020.34.S1.09512 |
0.307 |
|
2019 |
Bermudez D, Azad P, Figueroa-Mujíca R, Vizcardo-Galindo G, Corante N, Guerra-Giraldez C, Haddad GG, Villafuerte FC. Increased hypoxic proliferative response and gene expression in erythroid progenitor cells of Andean highlanders with Chronic Mountain Sickness. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 31617751 DOI: 10.1152/Ajpregu.00250.2019 |
0.316 |
|
2017 |
Stobdan T, Akbari A, Azad P, Zhou D, Poulsen O, Appenzeller O, Gonzales GF, Telenti A, Wong EHM, Saini S, Kirkness EF, Venter JC, Bafna V, Haddad GG. New insights into the genetic basis of Monge's disease and adaptation to high-altitude. Molecular Biology and Evolution. PMID 29029226 DOI: 10.1093/Molbev/Msx239 |
0.383 |
|
2017 |
Azad P, Stobdan T, Zhou D, Hartley I, Akbari A, Bafna V, Haddad GG. High-altitude adaptation in humans: from genomics to integrative physiology. Journal of Molecular Medicine (Berlin, Germany). PMID 28951950 DOI: 10.1007/S00109-017-1584-7 |
0.345 |
|
2016 |
Azad P, Zhao HW, Cabrales PJ, Ronen R, Zhou D, Poulsen O, Appenzeller O, Hsiao YH, Bafna V, Haddad GG. Senp1 drives hypoxia-induced polycythemia via GATA1 and Bcl-xL in subjects with Monge's disease. The Journal of Experimental Medicine. PMID 27821551 DOI: 10.1084/Jem.20151920 |
0.325 |
|
2014 |
Gersten M, Zhou D, Azad P, Haddad GG, Subramaniam S. Wnt pathway activation increases hypoxia tolerance during development. Plos One. 9: e103292. PMID 25093834 DOI: 10.1371/Journal.Pone.0103292 |
0.357 |
|
2012 |
Azad P, Zhou D, Zarndt R, Haddad GG. Identification of genes underlying hypoxia tolerance in Drosophila by a P-element screen. G3 (Bethesda, Md.). 2: 1169-78. PMID 23050227 DOI: 10.1534/G3.112.003681 |
0.34 |
|
2011 |
Azad P, Ryu J, Haddad GG. Distinct role of Hsp70 in Drosophila hemocytes during severe hypoxia. Free Radical Biology & Medicine. 51: 530-8. PMID 21616137 DOI: 10.1016/J.Freeradbiomed.2011.05.005 |
0.32 |
|
2011 |
Zhang M, Azad P, Woodruff RC. Adaptation of Drosophila melanogaster to increased NaCl concentration due to dominant beneficial mutations. Genetica. 139: 177-86. PMID 21128095 DOI: 10.1007/S10709-010-9535-Z |
0.558 |
|
2010 |
Azad P, Zhang M, Woodruff RC. Rapid increase in viability due to new beneficial mutations in Drosophila melanogaster. Genetica. 138: 251-63. PMID 19882309 DOI: 10.1007/S10709-009-9418-3 |
0.556 |
|
2009 |
Azad P, Haddad GG. Survival in acute and severe low o environment: use of a genetic model system. Annals of the New York Academy of Sciences. 1177: 39-47. PMID 19845605 DOI: 10.1111/J.1749-6632.2009.05045.X |
0.352 |
|
2009 |
Azad P, Zhou D, Russo E, Haddad GG. Distinct mechanisms underlying tolerance to intermittent and constant hypoxia in Drosophila melanogaster. Plos One. 4: e5371. PMID 19401761 DOI: 10.1371/Journal.Pone.0005371 |
0.317 |
|
2006 |
Azad P, Woodruff RC. Mutation and cloning efficiency. Cloning and Stem Cells. 8: 237-9. PMID 17196088 DOI: 10.1089/Clo.2006.8.237 |
0.518 |
|
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