Year |
Citation |
Score |
2018 |
Trease AJ, Li H, Spagnol G, Zheng L, Stauch KL, Sorgen PL. Regulation of Connexin32 by ephrin receptors and T-cell protein tyrosine phosphatase. The Journal of Biological Chemistry. PMID 30401746 DOI: 10.1074/jbc.RA118.003883 |
0.56 |
|
2016 |
Stauch KL, Villeneuve LM, Purnell PR, Ottemann BM, Emanuel K, Fox HS. Loss of Pink1 modulates synaptic mitochondrial bioenergetics in the rat striatum prior to motor symptoms: concomitant complex I respiratory defects and increased complex II-mediated respiration. Proteomics. Clinical Applications. PMID 27568932 DOI: 10.1002/prca.201600005 |
0.56 |
|
2016 |
Li H, Spagnol G, Zheng L, Stauch KL, Sorgen PL. Regulation of Connexin43 function and expression by Tyrosine kinase 2. The Journal of Biological Chemistry. PMID 27235399 DOI: 10.1074/jbc.M116.727008 |
0.56 |
|
2016 |
Ivanisevic J, Stauch KL, Petrascheck M, Benton HP, Epstein AA, Fang M, Gorantla S, Tran M, Hoang L, Kurczy ME, Boska MD, Gendelman HE, Fox HS, Siuzdak G. Metabolic drift in the aging brain. Aging. PMID 27182841 DOI: 10.18632/aging.100961 |
0.56 |
|
2016 |
Spagnol G, Kieken F, Kopanic JL, Li H, Zach S, Stauch KL, Grosely R, Sorgen PL. Structural Studies of the Nedd4 WW Domains and their Selectivity for the Cx43 Carboxyl-terminus. The Journal of Biological Chemistry. PMID 26841867 DOI: 10.1074/jbc.M115.701417 |
0.56 |
|
2016 |
Villeneuve LM, Purnell PR, Stauch KL, Fox HS. Neonatal mitochondrial abnormalities due to PINK1 deficiency: Proteomics reveals early changes relevant to Parkinson's disease Data in Brief. 6: 428-432. DOI: 10.1016/j.dib.2015.11.070 |
0.56 |
|
2016 |
Villeneuve LM, Purnell PR, Stauch KL, Callen SE, Buch SJ, Fox HS. HIV-1 transgenic rats display mitochondrial abnormalities consistent with abnormal energy generation and distribution Journal of Neurovirology. 1-11. DOI: 10.1007/s13365-016-0424-9 |
0.56 |
|
2015 |
Spagnol G, Al-Mugotir M, Kopanic JL, Zach S, Li H, Trease AJ, Stauch KL, Grosely R, Cervantes M, Sorgen PL. Secondary structural analysis of the carboxyl-terminal domain from different connexin isoforms. Biopolymers. PMID 26542351 DOI: 10.1002/bip.22762 |
0.56 |
|
2015 |
Stauch KL, Purnell PR, Villeneuve LM, Fox HS. Proteomic analysis and functional characterization of mouse brain mitochondria during aging reveal alterations in energy metabolism. Proteomics. 15: 1574-86. PMID 25546256 DOI: 10.1002/pmic.201400277 |
0.56 |
|
2015 |
Stauch KL, Purnell PR, Villeneuve LM, Fox HS. Data for mitochondrial proteomic alterations in the aging mouse brain Data in Brief. 4: 127-129. DOI: 10.1016/j.dib.2015.05.004 |
0.56 |
|
2014 |
Villeneuve LM, Stauch KL, Fox HS. Proteomic analysis of the mitochondria from embryonic and postnatal rat brains reveals response to developmental changes in energy demands. Journal of Proteomics. 109: 228-39. PMID 25046836 DOI: 10.1016/j.jprot.2014.07.011 |
0.56 |
|
2014 |
Stauch KL, Purnell PR, Fox HS. Aging synaptic mitochondria exhibit dynamic proteomic changes while maintaining bioenergetic function. Aging. 6: 320-34. PMID 24827396 DOI: 10.18632/aging.100657 |
0.56 |
|
2014 |
Stauch KL, Purnell PR, Fox HS. Quantitative proteomics of synaptic and nonsynaptic mitochondria: insights for synaptic mitochondrial vulnerability. Journal of Proteome Research. 13: 2620-36. PMID 24708184 DOI: 10.1021/pr500295n |
0.56 |
|
2014 |
Villeneuve LM, Stauch KL, Fox HS. Data for mitochondrial proteomic alterations in the developing rat brain Data in Brief. 1: 42-45. DOI: 10.1016/j.dib.2014.07.002 |
0.56 |
|
2014 |
Stauch KL, Fox HS. HIV/NeuroAIDS Neuroinflammation and Neurodegeneration. 247-262. DOI: 10.1007/978-1-4939-1071-7_12 |
0.56 |
|
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