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Antony S. Tin, Ph.D. - Publications

Affiliations: 
2013 Endocrinology University of California, Berkeley, Berkeley, CA, United States 
Area:
Molecular Biology, Cell Biology, Oncology
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5 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2014 Tin AS, Park AH, Sundar SN, Firestone GL. Essential role of the cancer stem/progenitor cell marker nucleostemin for indole-3-carbinol anti-proliferative responsiveness in human breast cancer cells. Bmc Biology. 12: 72. PMID 25209720 DOI: 10.1186/S12915-014-0072-6  0.601
2014 Tran KQ, Tin AS, Firestone GL. Artemisinin triggers a G1 cell cycle arrest of human Ishikawa endometrial cancer cells and inhibits cyclin-dependent kinase-4 promoter activity and expression by disrupting nuclear factor-κB transcriptional signaling. Anti-Cancer Drugs. 25: 270-81. PMID 24296733 DOI: 10.1097/Cad.0000000000000054  0.693
2012 Tin AS, Sundar SN, Tran KQ, Park AH, Poindexter KM, Firestone GL. Antiproliferative effects of artemisinin on human breast cancer cells requires the downregulated expression of the E2F1 transcription factor and loss of E2F1-target cell cycle genes. Anti-Cancer Drugs. 23: 370-9. PMID 22185819 DOI: 10.1097/Cad.0B013E32834F6Ea8  0.714
2011 Marconett CN, Sundar SN, Tseng M, Tin AS, Tran KQ, Mahuron KM, Bjeldanes LF, Firestone GL. Indole-3-carbinol downregulation of telomerase gene expression requires the inhibition of estrogen receptor-alpha and Sp1 transcription factor interactions within the hTERT promoter and mediates the G1 cell cycle arrest of human breast cancer cells. Carcinogenesis. 32: 1315-23. PMID 21693539 DOI: 10.1093/Carcin/Bgr116  0.612
2009 Willoughby JA, Sundar SN, Cheung M, Tin AS, Modiano J, Firestone GL. Artemisinin blocks prostate cancer growth and cell cycle progression by disrupting Sp1 interactions with the cyclin-dependent kinase-4 (CDK4) promoter and inhibiting CDK4 gene expression. The Journal of Biological Chemistry. 284: 2203-13. PMID 19017637 DOI: 10.1074/Jbc.M804491200  0.596
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