Richard R. Kew - Publications

Stony Brook University, Stony Brook, NY, United States 
Immunology, Molecular Biology, Cell Biology

19 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2016 Pednekar L, Valentino A, Ji Y, Tumma N, Valentino C, Kadoor A, Hosszu KK, Ramadass M, Kew RR, Kishore U, Peerschke EI, Ghebrehiwet B. Identification of the gC1qR sites for the HIV-1 viral envelope protein gp41 and the HCV core protein: Implications in viral-specific pathogenesis and therapy. Molecular Immunology. 74: 18-26. PMID 27111569 DOI: 10.1016/j.molimm.2016.03.016  0.68
2015 Ramadass M, Ghebrehiwet B, Kew RR. Enhanced recognition of plasma proteins in a non-native state by complement C3b. A possible clearance mechanism for damaged proteins in blood. Molecular Immunology. 64: 55-62. PMID 25466612 DOI: 10.1016/j.molimm.2014.10.022  0.68
2014 Penzo M, Habiel DM, Ramadass M, Kew RR, Marcu KB. Cell migration to CXCL12 requires simultaneous IKKα and IKKβ-dependent NF-κB signaling. Biochimica Et Biophysica Acta. 1843: 1796-804. PMID 24747690 DOI: 10.1016/j.bbamcr.2014.04.011  0.68
2014 Ramadass M, Ghebrehiwet B, Smith RJ, Kew RR. Generation of multiple fluid-phase C3b:plasma protein complexes during complement activation: possible implications in C3 glomerulopathies. Journal of Immunology (Baltimore, Md. : 1950). 192: 1220-30. PMID 24367026 DOI: 10.4049/jimmunol.1302288  0.68
2014 Ghebrehiwet B, Ji Y, Valentino A, Pednekar L, Ramadass M, Habiel D, Kew RR, Hosszu KH, Galanakis DK, Kishore U, Peerschke EI. Soluble gC1qR is an autocrine signal that induces B1R expression on endothelial cells. Journal of Immunology (Baltimore, Md. : 1950). 192: 377-84. PMID 24319267 DOI: 10.4049/jimmunol.1302031  0.68
2014 Ge L, Trujillo G, Miller EJ, Kew RR. Circulating complexes of the vitamin D binding protein with G-actin induce lung inflammation by targeting endothelial cells. Immunobiology. 219: 198-207. PMID 24268110 DOI: 10.1016/j.imbio.2013.10.001  0.68
2013 Trujillo G, Habiel DM, Ge L, Ramadass M, Cooke NE, Kew RR. Neutrophil recruitment to the lung in both C5a- and CXCL1-induced alveolitis is impaired in vitamin D-binding protein-deficient mice. Journal of Immunology (Baltimore, Md. : 1950). 191: 848-56. PMID 23752613 DOI: 10.4049/jimmunol.1202941  0.68
2012 Kew RR, Penzo M, Habiel DM, Marcu KB. The IKKα-dependent NF-κB p52/RelB noncanonical pathway is essential to sustain a CXCL12 autocrine loop in cells migrating in response to HMGB1. Journal of Immunology (Baltimore, Md. : 1950). 188: 2380-6. PMID 22287708 DOI: 10.4049/jimmunol.1102454  0.68
2011 Trujillo G, Zhang J, Habiel DM, Ge L, Ramadass M, Ghebrehiwet B, Kew RR. Cofactor regulation of C5a chemotactic activity in physiological fluids. Requirement for the vitamin D binding protein, thrombospondin-1 and its receptors. Molecular Immunology. 49: 495-503. PMID 22014686 DOI: 10.1016/j.molimm.2011.09.024  0.88
2010 Penzo M, Molteni R, Suda T, Samaniego S, Raucci A, Habiel DM, Miller F, Jiang HP, Li J, Pardi R, Palumbo R, Olivotto E, Kew RR, Bianchi ME, Marcu KB. Inhibitor of NF-kappa B kinases alpha and beta are both essential for high mobility group box 1-mediated chemotaxis [corrected]. Journal of Immunology (Baltimore, Md. : 1950). 184: 4497-509. PMID 20231695 DOI: 10.4049/jimmunol.0903131  0.68
2010 Zhang J, Habiel DM, Ramadass M, Kew RR. Identification of two distinct cell binding sequences in the vitamin D binding protein. Biochimica Et Biophysica Acta. 1803: 623-9. PMID 20211661 DOI: 10.1016/j.bbamcr.2010.02.010  0.68
2007 DiMartino SJ, Trujillo G, McVoy LA, Zhang J, Kew RR. Upregulation of vitamin D binding protein (Gc-globulin) binding sites during neutrophil activation from a latent reservoir in azurophil granules. Molecular Immunology. 44: 2370-7. PMID 17113648 DOI: 10.1016/j.molimm.2006.10.011  1
2006 Vegh Z, Kew RR, Gruber BL, Ghebrehiwet B. Chemotaxis of human monocyte-derived dendritic cells to complement component C1q is mediated by the receptors gC1qR and cC1qR. Molecular Immunology. 43: 1402-7. PMID 16140380 DOI: 10.1016/j.molimm.2005.07.030  0.44
2006 Shah AB, DiMartino SJ, Trujillo G, Kew RR. Selective inhibition of the C5a chemotactic cofactor function of the vitamin D binding protein by 1,25(OH)2 vitamin D3. Molecular Immunology. 43: 1109-15. PMID 16115686 DOI: 10.1016/j.molimm.2005.07.023  0.68
2005 McVoy LA, Kew RR. CD44 and annexin A2 mediate the C5a chemotactic cofactor function of the vitamin D binding protein. Journal of Immunology (Baltimore, Md. : 1950). 175: 4754-60. PMID 16177123  1
2004 Zhang J, Kew RR. Identification of a region in the vitamin D-binding protein that mediates its C5a chemotactic cofactor function. The Journal of Biological Chemistry. 279: 53282-7. PMID 15485893 DOI: 10.1074/jbc.M411462200  0.56
2004 Gruber BL, Marchese MJ, Santiago-Schwarz F, Martin CA, Zhang J, Kew RR. Protease-activated receptor-2 (PAR-2) expression in human fibroblasts is regulated by growth factors and extracellular matrix. The Journal of Investigative Dermatology. 123: 832-9. PMID 15482468 DOI: 10.1111/j.0022-202X.2004.23445.x  0.56
2004 Trujillo G, Kew RR. Platelet-derived thrombospondin-1 is necessary for the vitamin D-binding protein (Gc-globulin) to function as a chemotactic cofactor for C5a. Journal of Immunology (Baltimore, Md. : 1950). 173: 4130-6. PMID 15356163  0.88
2001 DiMartino SJ, Shah AB, Trujillo G, Kew RR. Elastase controls the binding of the vitamin D-binding protein (Gc-globulin) to neutrophils: a potential role in the regulation of C5a co-chemotactic activity. Journal of Immunology (Baltimore, Md. : 1950). 166: 2688-94. PMID 11160333  0.68
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