Qianghua Hu, Ph.D. - Publications
Affiliations: | 2006 | The University of Texas School of Public Health |
Area:
Molecular Biology, Cell BiologyYear | Citation | Score | |||
---|---|---|---|---|---|
2020 | Shen X, Wang R, Kim MJ, Hu Q, Hsu CC, Yao J, Klages-Mundt N, Tian Y, Lynn E, Brewer TF, Zhang Y, Arun B, Gan B, Andreeff M, Takeda S, et al. A Surge of DNA Damage Links Transcriptional Reprogramming and Hematopoietic Deficit in Fanconi Anemia. Molecular Cell. 80: 1013-1024.e6. PMID 33338401 DOI: 10.1016/j.molcel.2020.11.040 | 0.304 | |||
2008 | Luo R, Lu JF, Hu Q, Maity SN. CBF/NF-Y controls endoplasmic reticulum stress induced transcription through recruitment of both ATF6(N) and TBP. Journal of Cellular Biochemistry. 104: 1708-23. PMID 18348279 DOI: 10.1002/Jcb.21736 | 0.605 | |||
2006 | Hu Q, Lu JF, Luo R, Sen S, Maity SN. Inhibition of CBF/NF-Y mediated transcription activation arrests cells at G2/M phase and suppresses expression of genes activated at G2/M phase of the cell cycle. Nucleic Acids Research. 34: 6272-85. PMID 17098936 DOI: 10.1093/Nar/Gkl801 | 0.638 | |||
2002 | Hu Q, Bhattacharya C, Maity SN. CCAAT binding factor (CBF) binding mediates cell cycle activation of topoisomerase IIalpha. Conventional CBF activation domains are not required. The Journal of Biological Chemistry. 277: 37191-200. PMID 12149265 DOI: 10.1074/Jbc.M205985200 | 0.657 | |||
2001 | Coustry F, Hu Q, de Crombrugghe B, Maity SN. CBF/NF-Y functions both in nucleosomal disruption and transcription activation of the chromatin-assembled topoisomerase IIalpha promoter. Transcription activation by CBF/NF-Y in chromatin is dependent on the promoter structure. The Journal of Biological Chemistry. 276: 40621-30. PMID 11514576 DOI: 10.1074/Jbc.M106918200 | 0.653 | |||
2000 | Hu Q, Maity SN. Stable expression of a dominant negative mutant of CCAAT binding factor/NF-Y in mouse fibroblast cells resulting in retardation of cell growth and inhibition of transcription of various cellular genes. The Journal of Biological Chemistry. 275: 4435-44. PMID 10660616 DOI: 10.1074/Jbc.275.6.4435 | 0.639 | |||
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