Qianghua Hu, Ph.D. - Publications

Affiliations: 
2006 The University of Texas School of Public Health 
Area:
Molecular Biology, Cell Biology

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2020 Shen X, Wang R, Kim MJ, Hu Q, Hsu CC, Yao J, Klages-Mundt N, Tian Y, Lynn E, Brewer TF, Zhang Y, Arun B, Gan B, Andreeff M, Takeda S, et al. A Surge of DNA Damage Links Transcriptional Reprogramming and Hematopoietic Deficit in Fanconi Anemia. Molecular Cell. 80: 1013-1024.e6. PMID 33338401 DOI: 10.1016/j.molcel.2020.11.040  0.304
2008 Luo R, Lu JF, Hu Q, Maity SN. CBF/NF-Y controls endoplasmic reticulum stress induced transcription through recruitment of both ATF6(N) and TBP. Journal of Cellular Biochemistry. 104: 1708-23. PMID 18348279 DOI: 10.1002/Jcb.21736  0.605
2006 Hu Q, Lu JF, Luo R, Sen S, Maity SN. Inhibition of CBF/NF-Y mediated transcription activation arrests cells at G2/M phase and suppresses expression of genes activated at G2/M phase of the cell cycle. Nucleic Acids Research. 34: 6272-85. PMID 17098936 DOI: 10.1093/Nar/Gkl801  0.638
2002 Hu Q, Bhattacharya C, Maity SN. CCAAT binding factor (CBF) binding mediates cell cycle activation of topoisomerase IIalpha. Conventional CBF activation domains are not required. The Journal of Biological Chemistry. 277: 37191-200. PMID 12149265 DOI: 10.1074/Jbc.M205985200  0.657
2001 Coustry F, Hu Q, de Crombrugghe B, Maity SN. CBF/NF-Y functions both in nucleosomal disruption and transcription activation of the chromatin-assembled topoisomerase IIalpha promoter. Transcription activation by CBF/NF-Y in chromatin is dependent on the promoter structure. The Journal of Biological Chemistry. 276: 40621-30. PMID 11514576 DOI: 10.1074/Jbc.M106918200  0.653
2000 Hu Q, Maity SN. Stable expression of a dominant negative mutant of CCAAT binding factor/NF-Y in mouse fibroblast cells resulting in retardation of cell growth and inhibition of transcription of various cellular genes. The Journal of Biological Chemistry. 275: 4435-44. PMID 10660616 DOI: 10.1074/Jbc.275.6.4435  0.639
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