Deanna A. Mele, Ph.D. - Publications

2009 Cellular & Molecular Physiology Sackler School of Graduate Biomedical Sciences (Tufts University) 
Cell Biology, Molecular Biology

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any innacuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2013 Mele DA, Salmeron A, Ghosh S, Huang HR, Bryant BM, Lora JM. BET bromodomain inhibition suppresses TH17-mediated pathology. The Journal of Experimental Medicine. 210: 2181-90. PMID 24101376 DOI: 10.1084/jem.20130376  0.48
2011 Mertz JA, Conery AR, Bryant BM, Sandy P, Balasubramanian S, Mele DA, Bergeron L, Sims RJ. Targeting MYC dependence in cancer by inhibiting BET bromodomains. Proceedings of the National Academy of Sciences of the United States of America. 108: 16669-74. PMID 21949397 DOI: 10.1073/pnas.1108190108  0.48
2011 Mele DA, Sampson JF, Huber BT. Th17 differentiation is the default program for DPP2-deficient T-cell differentiation. European Journal of Immunology. 41: 1583-93. PMID 21469121 DOI: 10.1002/eji.201041157  0.48
2009 Danilova OV, Tai AK, Mele DA, Beinborn M, Leiter AB, Greenberg AS, Perfield JW, Defuria J, Singru PS, Lechan RM, Huber BT. Neurogenin 3-specific dipeptidyl peptidase-2 deficiency causes impaired glucose tolerance, insulin resistance, and visceral obesity. Endocrinology. 150: 5240-8. PMID 19819973 DOI: 10.1210/en.2009-0386  0.48
2009 Mele DA, Bista P, Baez DV, Huber BT. Dipeptidyl peptidase 2 is an essential survival factor in the regulation of cell quiescence. Cell Cycle (Georgetown, Tex.). 8: 2425-34. PMID 19556882  0.48
2008 Bista P, Mele DA, Baez DV, Huber BT. Lymphocyte quiescence factor Dpp2 is transcriptionally activated by KLF2 and TOB1. Molecular Immunology. 45: 3618-23. PMID 18555530 DOI: 10.1016/j.molimm.2008.05.001  0.48
Show low-probability matches.