Year |
Citation |
Score |
2016 |
Accardi MV, Troncy E, Abtout S, Ascah A, Maghezzi S, Authier S. Rat cardiovascular telemetry: Marginal distribution applied to positive control drugs. Journal of Pharmacological and Toxicological Methods. 81: 120-127. PMID 27039258 DOI: 10.1016/J.Vascn.2016.03.005 |
0.313 |
|
2011 |
Daussin FN, Godin R, Ascah A, Deschênes S, Burelle Y. Cyclophilin-D is dispensable for atrophy and mitochondrial apoptotic signalling in denervated muscle. The Journal of Physiology. 589: 855-61. PMID 21224232 DOI: 10.1113/Jphysiol.2010.202036 |
0.637 |
|
2011 |
Ascah A, Khairallah M, Daussin F, Bourcier-Lucas C, Godin R, Allen BG, Petrof BJ, Des Rosiers C, Burelle Y. Stress-induced opening of the permeability transition pore in the dystrophin-deficient heart is attenuated by acute treatment with sildenafil. American Journal of Physiology. Heart and Circulatory Physiology. 300: H144-53. PMID 20971771 DOI: 10.1152/Ajpheart.00522.2010 |
0.685 |
|
2010 |
Godin R, Ascah A, Daussin FN. Intensity-dependent activation of intracellular signalling pathways in skeletal muscle: Role of fibre type recruitment during exercise Journal of Physiology. 588: 4073-4074. PMID 21037317 DOI: 10.1113/Jphysiol.2010.195925 |
0.333 |
|
2010 |
Oliver-Dussault C, Ascah A, Marcil M, Matas J, Picard S, Pibarot P, Burelle Y, Deschepper CF. Early predictors of cardiac decompensation in experimental volume overload. Molecular and Cellular Biochemistry. 338: 271-82. PMID 20054615 DOI: 10.1007/S11010-009-0361-5 |
0.611 |
|
2010 |
Burelle Y, Khairallah M, Ascah A, Allen BG, Deschepper CF, Petrof BJ, Des Rosiers C. Alterations in mitochondrial function as a harbinger of cardiomyopathy: lessons from the dystrophic heart. Journal of Molecular and Cellular Cardiology. 48: 310-21. PMID 19769982 DOI: 10.1016/J.Yjmcc.2009.09.004 |
0.662 |
|
2009 |
Matas J, Young NT, Bourcier-Lucas C, Ascah A, Marcil M, Deschepper CF, Burelle Y. Increased expression and intramitochondrial translocation of cyclophilin-D associates with increased vulnerability of the permeability transition pore to stress-induced opening during compensated ventricular hypertrophy. Journal of Molecular and Cellular Cardiology. 46: 420-30. PMID 19094991 DOI: 10.1016/J.Yjmcc.2008.10.020 |
0.672 |
|
2008 |
Picard M, Csukly K, Robillard ME, Godin R, Ascah A, Bourcier-Lucas C, Burelle Y. Resistance to Ca2+-induced opening of the permeability transition pore differs in mitochondria from glycolytic and oxidative muscles. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 295: R659-68. PMID 18495829 DOI: 10.1152/Ajpregu.90357.2008 |
0.645 |
|
2006 |
Marcil M, Ascah A, Matas J, Bélanger S, Deschepper CF, Burelle Y. Compensated volume overload increases the vulnerability of heart mitochondria without affecting their functions in the absence of stress. Journal of Molecular and Cellular Cardiology. 41: 998-1009. PMID 17064727 DOI: 10.1016/J.Yjmcc.2006.08.117 |
0.689 |
|
2006 |
Ciminelli M, Ascah A, Bourduas K, Burelle Y. Short term training attenuates opening of the mitochondrial permeability transition pore without affecting myocardial function following ischemia-reperfusion. Molecular and Cellular Biochemistry. 291: 39-47. PMID 16718362 DOI: 10.1007/S11010-006-9192-9 |
0.652 |
|
2006 |
Csukly K, Ascah A, Matas J, Gardiner PF, Fontaine E, Burelle Y. Muscle denervation promotes opening of the permeability transition pore and increases the expression of cyclophilin D. The Journal of Physiology. 574: 319-27. PMID 16675492 DOI: 10.1113/Jphysiol.2006.109702 |
0.654 |
|
2006 |
Marcil M, Bourduas K, Ascah A, Burelle Y. Exercise training induces respiratory substrate-specific decrease in Ca2+-induced permeability transition pore opening in heart mitochondria. American Journal of Physiology. Heart and Circulatory Physiology. 290: H1549-57. PMID 16284229 DOI: 10.1152/Ajpheart.00913.2005 |
0.636 |
|
2006 |
Marcil M, Ascah A, Bélanger S, Deschepper C, Burelle Y. A52. Chronic volume overload increases the vulnerability of cardiac mitochondria without affecting their basal functions Journal of Molecular and Cellular Cardiology. 40: 865-866. DOI: 10.1016/J.Yjmcc.2006.03.289 |
0.611 |
|
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