Alexis Ascah, Ph.D. - Publications

Affiliations: 
2010 Université de Montréal, Montréal, Canada 
Area:
Pharmacology

13 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2016 Accardi MV, Troncy E, Abtout S, Ascah A, Maghezzi S, Authier S. Rat cardiovascular telemetry: Marginal distribution applied to positive control drugs. Journal of Pharmacological and Toxicological Methods. 81: 120-127. PMID 27039258 DOI: 10.1016/J.Vascn.2016.03.005  0.313
2011 Daussin FN, Godin R, Ascah A, Deschênes S, Burelle Y. Cyclophilin-D is dispensable for atrophy and mitochondrial apoptotic signalling in denervated muscle. The Journal of Physiology. 589: 855-61. PMID 21224232 DOI: 10.1113/Jphysiol.2010.202036  0.637
2011 Ascah A, Khairallah M, Daussin F, Bourcier-Lucas C, Godin R, Allen BG, Petrof BJ, Des Rosiers C, Burelle Y. Stress-induced opening of the permeability transition pore in the dystrophin-deficient heart is attenuated by acute treatment with sildenafil. American Journal of Physiology. Heart and Circulatory Physiology. 300: H144-53. PMID 20971771 DOI: 10.1152/Ajpheart.00522.2010  0.685
2010 Godin R, Ascah A, Daussin FN. Intensity-dependent activation of intracellular signalling pathways in skeletal muscle: Role of fibre type recruitment during exercise Journal of Physiology. 588: 4073-4074. PMID 21037317 DOI: 10.1113/Jphysiol.2010.195925  0.333
2010 Oliver-Dussault C, Ascah A, Marcil M, Matas J, Picard S, Pibarot P, Burelle Y, Deschepper CF. Early predictors of cardiac decompensation in experimental volume overload. Molecular and Cellular Biochemistry. 338: 271-82. PMID 20054615 DOI: 10.1007/S11010-009-0361-5  0.611
2010 Burelle Y, Khairallah M, Ascah A, Allen BG, Deschepper CF, Petrof BJ, Des Rosiers C. Alterations in mitochondrial function as a harbinger of cardiomyopathy: lessons from the dystrophic heart. Journal of Molecular and Cellular Cardiology. 48: 310-21. PMID 19769982 DOI: 10.1016/J.Yjmcc.2009.09.004  0.662
2009 Matas J, Young NT, Bourcier-Lucas C, Ascah A, Marcil M, Deschepper CF, Burelle Y. Increased expression and intramitochondrial translocation of cyclophilin-D associates with increased vulnerability of the permeability transition pore to stress-induced opening during compensated ventricular hypertrophy. Journal of Molecular and Cellular Cardiology. 46: 420-30. PMID 19094991 DOI: 10.1016/J.Yjmcc.2008.10.020  0.672
2008 Picard M, Csukly K, Robillard ME, Godin R, Ascah A, Bourcier-Lucas C, Burelle Y. Resistance to Ca2+-induced opening of the permeability transition pore differs in mitochondria from glycolytic and oxidative muscles. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 295: R659-68. PMID 18495829 DOI: 10.1152/Ajpregu.90357.2008  0.645
2006 Marcil M, Ascah A, Matas J, Bélanger S, Deschepper CF, Burelle Y. Compensated volume overload increases the vulnerability of heart mitochondria without affecting their functions in the absence of stress. Journal of Molecular and Cellular Cardiology. 41: 998-1009. PMID 17064727 DOI: 10.1016/J.Yjmcc.2006.08.117  0.689
2006 Ciminelli M, Ascah A, Bourduas K, Burelle Y. Short term training attenuates opening of the mitochondrial permeability transition pore without affecting myocardial function following ischemia-reperfusion. Molecular and Cellular Biochemistry. 291: 39-47. PMID 16718362 DOI: 10.1007/S11010-006-9192-9  0.652
2006 Csukly K, Ascah A, Matas J, Gardiner PF, Fontaine E, Burelle Y. Muscle denervation promotes opening of the permeability transition pore and increases the expression of cyclophilin D. The Journal of Physiology. 574: 319-27. PMID 16675492 DOI: 10.1113/Jphysiol.2006.109702  0.654
2006 Marcil M, Bourduas K, Ascah A, Burelle Y. Exercise training induces respiratory substrate-specific decrease in Ca2+-induced permeability transition pore opening in heart mitochondria. American Journal of Physiology. Heart and Circulatory Physiology. 290: H1549-57. PMID 16284229 DOI: 10.1152/Ajpheart.00913.2005  0.636
2006 Marcil M, Ascah A, Bélanger S, Deschepper C, Burelle Y. A52. Chronic volume overload increases the vulnerability of cardiac mitochondria without affecting their basal functions Journal of Molecular and Cellular Cardiology. 40: 865-866. DOI: 10.1016/J.Yjmcc.2006.03.289  0.611
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