Pierre Mazot - Publications

Affiliations: 
2012-2016 Icahn School of Medicine at Mount Sinai, New York, NY, United States 
 2017- INSERM - France 

14 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2021 Ray AT, Mazot P, Brewer JR, Catela C, Dinsmore CJ, Soriano P. Corrigendum: FGF signaling regulates development by processes beyond canonical pathways. Genes & Development. 35: 783. PMID 33941542 DOI: 10.1101/gad.348503.121  0.689
2020 Ray AT, Mazot P, Brewer JR, Catela C, Dinsmore CJ, Soriano P. FGF signaling regulates development by processes beyond canonical pathways. Genes & Development. PMID 33184218 DOI: 10.1101/gad.342956.120  0.716
2017 Molotkov A, Mazot P, Brewer JR, Cinalli RM, Soriano P. Distinct Requirements for FGFR1 and FGFR2 in Primitive Endoderm Development and Exit from Pluripotency. Developmental Cell. PMID 28552557 DOI: 10.1016/J.Devcel.2017.05.004  0.758
2016 Brewer JR, Mazot P, Soriano P. Genetic insights into the mechanisms of Fgf signaling. Genes & Development. 30: 751-71. PMID 27036966 DOI: 10.1101/Gad.277137.115  0.814
2015 Brewer JR, Molotkov A, Mazot P, Hoch RV, Soriano P. Fgfr1 regulates development through the combinatorial use of signaling proteins Genes & Development. 29: 1863-1874. PMID 26341559 DOI: 10.1101/Gad.264994.115  0.796
2015 Vasudevan HN, Mazot P, He F, Soriano P. Receptor tyrosine kinases modulate distinct transcriptional programs by differential usage of intracellular pathways. Elife. 4. PMID 25951516 DOI: 10.7554/Elife.07186  0.82
2015 Vasudevan HN, Mazot P, He F, Soriano P. Author response: Receptor tyrosine kinases modulate distinct transcriptional programs by differential usage of intracellular pathways Elife. DOI: 10.7554/Elife.07186.020  0.777
2015 Vasudevan HN, Mazot P, He F, Soriano P. Receptor tyrosine kinases modulate distinct transcriptional programs by differential usage of intracellular pathways Elife. 4. DOI: 10.7554/eLife.07186  0.785
2013 Boutterin MC, Mazot P, Faure C, Doly S, Gervasi N, Tremblay ML, Vigny M. Control of ALK (wild type and mutated forms) phosphorylation: specific role of the phosphatase PTP1B. Cellular Signalling. 25: 1505-13. PMID 23499906 DOI: 10.1016/j.cellsig.2013.02.020  0.362
2013 Cazes A, Louis-Brennetot C, Mazot P, Dingli F, Lombard B, Boeva V, Daveau R, Cappo J, Combaret V, Schleiermacher G, Jouannet S, Ferrand S, Pierron G, Barillot E, Loew D, et al. Characterization of rearrangements involving the ALK gene reveals a novel truncated form associated with tumor aggressiveness in neuroblastoma. Cancer Research. 73: 195-204. PMID 23139213 DOI: 10.1158/0008-5472.Can-12-1242  0.324
2012 Mazot P, Cazes A, Dingli F, Degoutin J, Irinopoulou T, Boutterin MC, Lombard B, Loew D, Hallberg B, Palmer RH, Delattre O, Janoueix-Lerosey I, Vigny M. Internalization and down-regulation of the ALK receptor in neuroblastoma cell lines upon monoclonal antibodies treatment. Plos One. 7: e33581. PMID 22479414 DOI: 10.1371/Journal.Pone.0033581  0.457
2011 Mazot P, Cazes A, Boutterin MC, Figueiredo A, Raynal V, Combaret V, Hallberg B, Palmer RH, Delattre O, Janoueix-Lerosey I, Vigny M. The constitutive activity of the ALK mutated at positions F1174 or R1275 impairs receptor trafficking. Oncogene. 30: 2017-25. PMID 21242967 DOI: 10.1038/Onc.2010.595  0.408
2010 Bennasroune A, Mazot P, Boutterin MC, Vigny M. Activation of the orphan receptor tyrosine kinase ALK by zinc. Biochemical and Biophysical Research Communications. 398: 702-6. PMID 20621063 DOI: 10.1016/j.bbrc.2010.07.004  0.309
2007 Mathivet T, Mazot P, Vigny M. In contrast to agonist monoclonal antibodies, both C-terminal truncated form and full length form of Pleiotrophin failed to activate vertebrate ALK (anaplastic lymphoma kinase)? Cellular Signalling. 19: 2434-43. PMID 17904822 DOI: 10.1016/j.cellsig.2007.07.011  0.324
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