Parents

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Halvor Orin Halvorson grad student 1949 UMN (ID Tree)
 (Co-advisor)
Sol Spiegelman grad student 1949 UMN
 (The dissociation of biosynthesis from glycolysis in the intact yeast cell)

Children

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Richard H. Kessin grad student Brandeis (Microtree)
S. Gaylen Bradley grad student 1954 Northwestern (Neurotree)
David R. Soll post-doc (Microtree)
BETA: Related publications

Publications

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Sin J, Puccini JM, Huang C, et al. (2016) Correction: The Impact of Juvenile Coxsackievirus Infection on Cardiac Progenitor Cells and Postnatal Heart Development. Plos Pathogens. 12: e1005673
Castaldi A, Chesini GP, Taylor AE, et al. (2016) Sphingosine 1-phosphate elicits RhoA-dependent proliferation and MRTF-A mediated gene induction in CPCs. Cellular Signalling
Broughton KM, Sussman MA. (2016) Empowering Adult Stem Cells for Myocardial Regeneration V2.0: Success in Small Steps. Circulation Research. 118: 867-80
Yokoi K, Onishi R, Deng XL, et al. (2016) Density-Scaled Balanced Continuum Surface Force Model with a Level Set Based Curvature Interpolation Technique International Journal of Computational Methods
Samse K, Hariharan N, Sussman MA. (2015) Personalizing cardiac regenerative therapy: at the heart of Pim1 kinase. Pharmacological Research
Quijada P, Hariharan N, Cubillo JD, et al. (2015) Nuclear Calcium/Calmodulin-Dependent Protein Kinase II Signaling Enhances Cardiac Progenitor Cell Survival and Cardiac Lineage Commitment. The Journal of Biological Chemistry
Quijada P, Salunga HT, Hariharan N, et al. (2015) Cardiac Stem Cell Hybrids Enhance Myocardial Repair. Circulation Research
Wade F, Quijada P, Al-Haffar KM, et al. (2015) Deletion of low molecular weight protein tyrosine phosphatase (Acp1) protects against stress-induced cardiomyopathy. The Journal of Pathology
Orogo AM, Gonzalez ER, Kubli DA, et al. (2015) Accumulation of Mitochondrial DNA Mutations Disrupts Cardiac Progenitor Cell Function and Reduces Survival. The Journal of Biological Chemistry
Doroudgar S, Völkers M, Thuerauf DJ, et al. (2015) Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes. Circulation Research. 117: 536-46
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