Jen-Tsan A. Chi, Ph.D.
Affiliations: | Stanford University, Palo Alto, CA |
Area:
Immunology, Molecular BiologyGoogle:
"Jen-Tsan Chi"Mean distance: 9.53 | S | N | B | C | P |
Parents
Sign in to add mentorMark M. Davis | grad student | 2000 | Stanford | |
(The mechanism of Blimp -1 in B cell terminal differentiation.) |
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Publications
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Kovach AR, Oristian KM, Kirsch DG, et al. (2022) Identification and targeting of a HES1-YAP1-CDKN1C functional interaction in fusion-negative rhabdomyosarcoma. Molecular Oncology |
Sun T, Chi J. (2020) Regulation of ferroptosis in cancer cells by YAP/TAZ and Hippo pathways: The therapeutic implications Genes and Diseases |
Yang W, Chi J. (2019) Abstract 4472: The hippo pathway effector TAZ regulates ferroptosis in renal cell carcinoma Cancer Research. 79: 4472-4472 |
Sun T, Ding C, Chi JA. (2019) Abstract 2667: Genetic removal of metazoan SpoT homolog I (MESH1) inhibits proliferation through the repression of HIPPO effector TAZ Cancer Research. 79: 2667-2667 |
Siamakpour-Reihani S, Chen W, Corcoran D, et al. (2016) Abstract 1647: Radiation response genome-wide analysis using paired pre and post-radiation FFPE human breast tumor samples Cancer Research. 76: 1647-1647 |
Moon EJ, Mello SS, Chi J, et al. (2016) Abstract 1628: MAFF, a new hypoxia target gene involving tumor invasion and metastasis Cancer Research. 76: 1628-1628 |
Horton JK, Siamakpour-Reihani S, Lee CT, et al. (2015) FAS Death Receptor: A Breast Cancer Subtype-Specific Radiation Response Biomarker and Potential Therapeutic Target. Radiation Research |
Siamakpour-Reihani S, Chen W, Lee C, et al. (2015) Abstract 3331: Gene expression profiling after radiation in human breast cancer specimens and breast cancer cell lines Cancer Research. 75: 3331-3331 |
Keenan MM, Liu B, Tang X, et al. (2015) Abstract 3004: Contextual RNAi screen identifies ACLY and ACC1 as mediators of hypoxia-induced apoptosis through metabolic and transcriptional mechanisms Cancer Research. 75: 3004-3004 |
Ding CC, Tang X, Kim SY, et al. (2015) Abstract 1100: Functional genomics to investigate the genetic determinants of cell death induced by oxidative stresses Cancer Research. 75: 1100-1100 |