Kenneth A. Myers, Ph.D.
Affiliations: | 2007 | Drexel University College of Medicine, Philadelphia, PA, United States |
Area:
Neuronal cytoskeletonGoogle:
"Kenneth Myers"Mean distance: 19.83 (cluster 52) | S | N | B | C | P |
Parents
Sign in to add mentor| Peter W. Baas | grad student | 2007 | Drexel Medical School | |
| (Axonal growth, retraction, and growth -cone navigation are regulated by the balance of molecular motor forces within the axon.) | ||||
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Publications
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| Myer NM, Myers KA. (2017) CLASP1 regulates endothelial cell branching morphology and directed migration. Biology Open |
| Rao AN, Patil A, Black MM, et al. (2017) Cytoplasmic Dynein Transports Axonal Microtubules in a Polarity-Sorting Manner. Cell Reports. 19: 2210-2219 |
| D'Angelo L, Myer NM, Myers KA. (2017) MCAK-mediated regulation of endothelial cell microtubule dynamics is mechano-sensitive to myosin-II contractility. Molecular Biology of the Cell |
| Braun A, Caesar NM, Dang K, et al. (2016) High-resolution Time-lapse Imaging and Automated Analysis of Microtubule Dynamics in Living Human Umbilical Vein Endothelial Cells. Journal of Visualized Experiments : Jove |
| Braun A, Dang K, Buslig F, et al. (2014) Rac1 and Aurora A regulate MCAK to polarize microtubule growth in migrating endothelial cells. The Journal of Cell Biology. 206: 97-112 |
| Baas PW, Myers KA, Qiang L, et al. (2014) Microtubules: Organization and Function in Neurons Reference Module in Biomedical Research |
| Myers KA, Applegate KT, Danuser G, et al. (2011) Distinct ECM mechanosensing pathways regulate microtubule dynamics to control endothelial cell branching morphogenesis. The Journal of Cell Biology. 192: 321-34 |
| Nadar VC, Ketschek A, Myers KA, et al. (2008) Kinesin-5 is essential for growth-cone turning. Current Biology : Cb. 18: 1972-7 |
| Myers KA, Baas PW. (2007) Kinesin-5 regulates the growth of the axon by acting as a brake on its microtubule array. The Journal of Cell Biology. 178: 1081-91 |
| Myers KA, Tint I, Nadar CV, et al. (2006) Antagonistic forces generated by cytoplasmic dynein and myosin-II during growth cone turning and axonal retraction. Traffic (Copenhagen, Denmark). 7: 1333-51 |