Bernhard U. Keller

Affiliations: 
Georg-August-Universität Göttingen, Göttingen, Niedersachsen, Germany 
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"Bernhard Keller"
Mean distance: 13.56 (cluster 32)
 
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Publications

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Fuchs A, Kutterer S, Mühling T, et al. (2013) Selective mitochondrial Ca2+ uptake deficit in disease endstage vulnerable motoneurons of the SOD1G93A mouse model of amyotrophic lateral sclerosis. The Journal of Physiology. 591: 2723-45
Grosskreutz J, Van Den Bosch L, Keller BU. (2010) Calcium dysregulation in amyotrophic lateral sclerosis. Cell Calcium. 47: 165-74
Jaiswal MK, Zech WD, Goos M, et al. (2009) Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease. Bmc Neuroscience. 10: 64
Jaiswal MK, Keller BU. (2009) Cu/Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of mitochondria and perturbs Ca2+ homeostasis in SOD1G93A mice. Molecular Pharmacology. 75: 478-89
von Lewinski F, Fuchs J, Vanselow BK, et al. (2008) Low Ca2+ buffering in hypoglossal motoneurons of mutant SOD1 (G93A) mice. Neuroscience Letters. 445: 224-8
Goos M, Zech WD, Jaiswal MK, et al. (2007) Expression of a Cu,Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of neuroblastoma cells to infectious injury. Bmc Infectious Diseases. 7: 131
Renner U, Glebov K, Lang T, et al. (2007) Localization of the mouse 5-hydroxytryptamine(1A) receptor in lipid microdomains depends on its palmitoylation and is involved in receptor-mediated signaling. Molecular Pharmacology. 72: 502-13
von Lewinski F, Keller BU. (2005) Ca2+, mitochondria and selective motoneuron vulnerability: implications for ALS. Trends in Neurosciences. 28: 494-500
von Lewinski F, Keller BU. (2005) Mitochondrial Ca2+ buffering in hypoglossal motoneurons from mouse. Neuroscience Letters. 380: 203-8
Ladewig T, Lalley PM, Keller BU. (2004) Serotonergic modulation of intracellular calcium dynamics in neonatal hypoglossal motoneurons from mouse. Brain Research. 1001: 1-12
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