Xinxin Yang, Ph.D.
Affiliations: | 2009 | Stony Brook University, Stony Brook, NY, United States |
Area:
proteins, chemical biology, Mtb, polymersGoogle:
"Xinxin Yang"Mean distance: 8.45 | S | N | B | C | P |
Parents
Sign in to add mentorNicole S. Sampson | grad student | 2009 | SUNY Stony Brook | |
(Characterization of 3beta-hydroxysteroid dehydrogenase (Rv1106c) and the sterol metabolic pathway in Mycobacterium tuberculosis (M. tb).) |
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Publications
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Chen YC, Yang X, Wang N, et al. (2023) Uncovering the Roles of in Redox and Bioenergetic Homeostasis: Implications for Antitubercular Therapy. Biorxiv : the Preprint Server For Biology |
Werman JM, Chen YC, Yuan T, et al. (2023) A Chemoproteomic Approach to Elucidate the Mechanism of Action of 6-Azasteroids with Unique Activity in Mycobacteria. Acs Infectious Diseases. 9: 1993-2004 |
Yang X, Wipperman MF, Nachman S, et al. (2022) Exploring the value of Mycobacterium tuberculosis modified lipoprotein as a potential biomarker for TB detection in children. Bmc Infectious Diseases. 22: 158 |
Gadbery J, Round J, Yuan T, et al. (2020) IpdE1-IpdE2 is a heterotetrameric acyl coenzyme A dehydrogenase that is widely distributed in steroid-degrading bacteria. Biochemistry |
Yang X, Yuan T, Ma R, et al. (2019) The Mce3R stress-resistance pathway is vulnerable to small molecule targeting that improves tuberculosis drug activities. Acs Infectious Diseases |
Zhao Q, Hu J, Xiang J, et al. (2015) Intranasal administration of human umbilical cord mesenchymal stem cells-conditioned medium enhances vascular remodeling after stroke. Brain Research. 1624: 489-96 |
Thomas ST, Yang X, Sampson NS. (2011) Inhibition of the M. tuberculosis 3β-hydroxysteroid dehydrogenase by azasteroids. Bioorganic & Medicinal Chemistry Letters. 21: 2216-9 |
Yang X, Gao J, Smith I, et al. (2011) Cholesterol is not an essential source of nutrition for Mycobacterium tuberculosis during infection. Journal of Bacteriology. 193: 1473-6 |
Nesbitt NM, Yang X, Fontán P, et al. (2010) A thiolase of Mycobacterium tuberculosis is required for virulence and production of androstenedione and androstadienedione from cholesterol. Infection and Immunity. 78: 275-82 |
Yang X, Nesbitt NM, Dubnau E, et al. (2009) Cholesterol metabolism increases the metabolic pool of propionate in Mycobacterium tuberculosis. Biochemistry. 48: 3819-21 |