Pasi Tavi

Affiliations: 
University of Kuopio, Kuopio, Finland 
Area:
cardiomyocyte physiology
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"Pasi Tavi"
Mean distance: 16.67 (cluster 6)
 
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Publications

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Kärkkäinen O, Tuomainen T, Mutikainen M, et al. (2018) Heart specific PGC-1α deletion identifies metabolome of cardiac restricted metabolic heart failure. Cardiovascular Research
Koivumäki JT, Naumenko N, Tuomainen T, et al. (2018) Structural Immaturity of Human iPSC-Derived Cardiomyocytes:Investigation of Effects on Function and Disease Modeling. Frontiers in Physiology. 9: 80
Karppinen S, Hänninen SL, Rapila R, et al. (2018) Sarcoplasmic reticulum Ca2+ -induced Ca2+ release regulates class IIa HDAC localization in mouse embryonic cardiomyocytes. Physiological Reports. 6
Winther SV, Tuomainen T, Borup R, et al. (2016) Potassium Channel Interacting Protein 2 (KChIP2) is not a transcriptional regulator of cardiac electrical remodeling. Scientific Reports. 6: 28760
Louch WE, Koivumäki JT, Tavi P. (2015) Calcium signalling in developing cardiomyocytes: implications for model systems and disease. The Journal of Physiology. 593: 1047-63
Koivumäki JT, Seemann G, Maleckar MM, et al. (2014) In silico screening of the key cellular remodeling targets in chronic atrial fibrillation. Plos Computational Biology. 10: e1003620
Ronkainen VP, Tuomainen T, Huusko J, et al. (2014) Hypoxia-inducible factor 1-induced G protein-coupled receptor 35 expression is an early marker of progressive cardiac remodelling. Cardiovascular Research. 101: 69-77
Ronkainen JJ, Hänninen SL, Korhonen T, et al. (2011) Ca2+-calmodulin-dependent protein kinase II represses cardiac transcription of the L-type calcium channel alpha(1C)-subunit gene (Cacna1c) by DREAM translocation. The Journal of Physiology. 589: 2669-86
Ronkainen VP, Skoumal R, Tavi P. (2011) Hypoxia and HIF-1 suppress SERCA2a expression in embryonic cardiac myocytes through two interdependent hypoxia response elements. Journal of Molecular and Cellular Cardiology. 50: 1008-16
Kubin AM, Skoumal R, Tavi P, et al. (2011) Role of reactive oxygen species in the regulation of cardiac contractility. Journal of Molecular and Cellular Cardiology. 50: 884-93
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