Kenneth E. Bernstein
Affiliations: | Emory University, Atlanta, GA |
Area:
Animal Physiology Biology, BiochemistryGoogle:
"Kenneth Bernstein"Mean distance: (not calculated yet)
Children
Sign in to add traineeJustin M. Cole | grad student | 2002 | Emory |
Derick Okwan-Duodu | grad student | 2013 | Emory |
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Publications
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Saito S, Cao DY, Bernstein EA, et al. (2025) Peroxisome proliferator-activated receptor alpha is an essential factor in enhanced macrophage immune function induced by angiotensin-converting enzyme. Cellular & Molecular Immunology |
Oosthuizen D, Ganief TA, Bernstein KE, et al. (2024) Proteomic Analysis of Human Macrophages Overexpressing Angiotensin-Converting Enzyme. International Journal of Molecular Sciences. 25 |
Bernstein KE, Cao D, Shibata T, et al. (2024) Classical and non-classical effects of angiotensin converting enzyme: how increased ACE enhances myeloid immune function. The Journal of Biological Chemistry. 107388 |
Saito S, Cao D, Bernstein EA, et al. (2024) Peroxisome proliferator-activated receptor alpha is essential factor in enhanced macrophage immune function induced by angiotensin converting enzyme. Research Square |
Gomez AR, Byun HR, Wu S, et al. (2024) Angiotensin Converting Enzyme (ACE) expression in microglia reduces amyloid β deposition and neurodegeneration by increasing SYK signaling and endolysosomal trafficking. Biorxiv : the Preprint Server For Biology |
Cao D, Saito S, Xu L, et al. (2023) Myeloid cell ACE shapes cellular metabolism and function in PCSK-9 induced atherosclerosis. Frontiers in Immunology. 14: 1278383 |
Cao D, Khan Z, Li X, et al. (2023) Macrophage angiotensin-converting enzyme (ACE) reduces atherosclerosis by increasing PPARα and fundamentally changing lipid metabolism. Cardiovascular Research |
Suhail H, Peng H, Xu J, et al. (2022) Knockout of ACE-N facilitates improved cardiac function after myocardial infarction. Journal of Molecular and Cellular Cardiology Plus. 3 |
Saito S, Tatsumoto N, Cao DY, et al. (2022) Overexpressed Angiotensin Converting Enzyme in Neutrophils Suppresses Glomerular Damage in Crescentic Glomerulonephritis. American Journal of Physiology. Renal Physiology |
Veiras LC, Bernstein EA, Cao D, et al. (2022) Tubular IL-1β Induces Salt Sensitivity in Diabetes by Activating Renal Macrophages. Circulation Research. 101161CIRCRESAHA1213 |