Wanli Tang, Ph.D.
Affiliations: | 2010 | Molecular Cancer Biology | Duke University, Durham, NC |
Area:
Molecular Biology, Cell BiologyGoogle:
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Parents
Sign in to add mentorSally Kornbluth | grad student | 2010 | Duke | |
(Regulation of anaphase promoting complex/cyclosome to control M phase exit.) |
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Publications
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Zhang L, Chen C, Huang N, et al. (2014) Abstract 2480: Ubiquitination of p53 by Trim39 Cancer Research. 74: 2480-2480 |
Zhang L, Huang NJ, Chen C, et al. (2012) Ubiquitylation of p53 by the APC/C inhibitor Trim39. Proceedings of the National Academy of Sciences of the United States of America. 109: 20931-6 |
Huang NJ, Zhang L, Tang W, et al. (2012) The Trim39 ubiquitin ligase inhibits APC/CCdh1-mediated degradation of the Bax activator MOAP-1. The Journal of Cell Biology. 197: 361-7 |
Horn SR, Thomenius MJ, Johnson ES, et al. (2011) Regulation of mitochondrial morphology by APC/CCdh1-mediated control of Drp1 stability. Molecular Biology of the Cell. 22: 1207-16 |
Yang CS, Thomenius MJ, Gan EC, et al. (2010) Metabolic regulation of Drosophila apoptosis through inhibitory phosphorylation of Dronc. The Embo Journal. 29: 3196-207 |
Tang W, Wu JQ, Chen C, et al. (2010) Emi2-mediated inhibition of E2-substrate ubiquitin transfer by the anaphase-promoting complex/cyclosome through a D-box-independent mechanism. Molecular Biology of the Cell. 21: 2589-97 |
Nutt LK, Buchakjian MR, Gan E, et al. (2010) Metabolic Control of Oocyte Apoptosis Mediated by 14-3-3ζ-Regulated Dephosphorylation of Caspase-2 (DOI:10.1016/j.devcel.2009.04.005) Developmental Cell. 18: 165 |
Nutt LK, Buchakjian MR, Gan E, et al. (2009) Metabolic control of oocyte apoptosis mediated by 14-3-3zeta-regulated dephosphorylation of caspase-2. Developmental Cell. 16: 856-66 |
Wu JQ, Guo JY, Tang W, et al. (2009) PP1-mediated dephosphorylation of phosphoproteins at mitotic exit is controlled by inhibitor-1 and PP1 phosphorylation. Nature Cell Biology. 11: 644-51 |
Guo JY, Yamada A, Kajino T, et al. (2008) Aven-dependent activation of ATM following DNA damage. Current Biology : Cb. 18: 933-42 |