Siu-Pok Yee
Affiliations: | The University of Western Ontario (Canada) |
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Children
Sign in to add traineePeter E. Bialek | grad student | 2001 | The University of Western Ontario (Canada) |
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Publications
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Man Y, Li W, Yap YT, et al. (2023) Generation of floxed Spag6l mice and disruption of the gene by crossing to a Hprt-Cre line. Genesis (New York, N.Y. : 2000). e23512 |
Baena V, Owen CM, Uliasz TF, et al. (2020) Cellular heterogeneity of the LH receptor and its significance for cyclic GMP signaling in mouse preovulatory follicles. Endocrinology |
Oakie A, Feng ZC, Li J, et al. (2019) Long-term c-Kit overexpression in beta cells compromises their function in ageing mice. Diabetologia |
Thirunavukkarasu M, Selvaraju V, Joshi M, et al. (2018) Disruption of VEGF Mediated Flk-1 Signaling Leads to a Gradual Loss of Vessel Health and Cardiac Function During Myocardial Infarction: Potential Therapy With Pellino-1. Journal of the American Heart Association. 7: e007601 |
Canalis E, Yu J, Schilling L, et al. (2018) The lateral meningocele syndrome mutation causes marked osteopenia in mice. The Journal of Biological Chemistry |
Egbert JR, Yee SP, Jaffe LA. (2018) Luteinizing hormone signaling phosphorylates and activates the cyclic GMP phosphodiesterase PDE5 in mouse ovarian follicles, contributing an additional component to the hormonally induced decrease in cyclic GMP that reinitiates meiosis. Developmental Biology |
Park JW, Yan L, Stoddard C, et al. (2017) Recapitulating and Correcting Marfan Syndrome in a Cellular Model. International Journal of Biological Sciences. 13: 588-603 |
Clark NC, Pru CA, Yee SP, et al. (2016) Conditional Ablation of Progesterone Receptor Membrane Component 2 Causes Female Premature Reproductive Senescence. Endocrinology. en20161701 |
McCallum ML, Pru CA, Niikura Y, et al. (2016) Conditional Ablation of Progesterone Receptor Membrane Component 1 Results in Subfertility in the Female and Development of Endometrial Cysts. Endocrinology. en20161081 |
Enkhmnandakh B, Stoddard C, Mack K, et al. (2016) Generation of a mouse model for a conditional inactivation of Gtf2i allele. Genesis (New York, N.Y. : 2000) |