Martin Friedlander
Affiliations: | Scripps Research Institute, La Jolla, La Jolla, CA, United States |
Area:
Cell Biology, Molecular Biology, BiochemistryGoogle:
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Publications
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Marra KV, Chen JS, Robles-Holmes HK, et al. (2023) Development of a Semi-automated Computer-based Tool for the Quantification of Vascular Tortuosity in the Murine Retina. Ophthalmology Science. 4: 100439 |
Chen JS, Marra KV, Robles-Holmes HK, et al. (2023) Applications of Deep Learning: Automated Assessment of Vascular Tortuosity in Mouse Models of Oxygen-Induced Retinopathy. Ophthalmology Science. 4: 100338 |
Rosarda JD, Giles S, Harkins-Perry S, et al. (2023) Imbalanced unfolded protein response signaling contributes to 1-deoxysphingolipid retinal toxicity. Nature Communications. 14: 4119 |
Usui-Ouchi A, Giles S, Harkins-Perry S, et al. (2023) Integrating human iPSC-derived macrophage progenitors into retinal organoids to generate a mature retinal microglial niche. Glia |
Eade KT, Ansell BRE, Giles S, et al. (2023) iPSC-derived retinal pigmented epithelial cells from patients with macular telangiectasia show decreased mitochondrial function. The Journal of Clinical Investigation. 133 |
Marra KV, Aguilar E, Guoqin W, et al. (2022) Bioactive extracellular vesicles from a subset of endothelial progenitor cells rescue retinal ischemia and neurodegeneration. Jci Insight |
Usui-Ouchi A, Eade K, Giles S, et al. (2022) Deletion of Tgfβ signal in activated microglia prolongs hypoxia-induced retinal neovascularization enhancing Igf1 expression and retinal leukostasis. Glia |
Thomas ED, Timms AE, Giles S, et al. (2022) Cell-specific cis-regulatory elements and mechanisms of non-coding genetic disease in human retina and retinal organoids. Developmental Cell |
Jung I, Jung D, Zha Z, et al. (2021) Interferon-γ inhibits retinal neovascularization in a mouse model of ischemic retinopathy. Cytokine. 155542 |
Eade K, Gantner ML, Hostyk JA, et al. (2021) Serine biosynthesis defect due to haploinsufficiency of PHGDH causes retinal disease. Nature Metabolism. 3: 366-377 |