Owen J. McCarty, Ph.D.

Affiliations: 
2003 Johns Hopkins University, Baltimore, MD 
Area:
Chemical Engineering, Oncology, Animal Physiology Biology, Immunology
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"Owen McCarty"
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Parents

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Konstantinos Konstantopoulos grad student 2003 Johns Hopkins
 (Flow modulation of receptor -mediated interactions in inflammation and cancer metastasis.)
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Publications

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Key NS, Khorana AA, Mackman N, et al. (2016) Thrombosis in Cancer: Research Priorities Identified by a National Cancer Institute/National Heart, Lung, and Blood Institute Strategic Working Group. Cancer Research. 76: 3671-3675
Healy LD, Puy C, Itakura A, et al. (2016) Colocalization of neutrophils, extracellular DNA and coagulation factors during NETosis: Development and utility of an immunofluorescence-based microscopy platform. Journal of Immunological Methods
Bane CE, Ivanov I, Matafonov A, et al. (2016) Factor XI Deficiency Alters the Cytokine Response and Activation of Contact Proteases during Polymicrobial Sepsis in Mice. Plos One. 11: e0152968
Baker-Groberg SM, Lattimore S, Recht M, et al. (2016) Assessment of neonatal platelet adhesion, activation, and aggregation. Journal of Thrombosis and Haemostasis : Jth
Aslan JE, David LL, McCarty OJ. (2015) Data detailing the platelet acetyl-lysine proteome. Data in Brief. 5: 368-71
Mattheij NJ, Swieringa F, Mastenbroek TG, et al. (2015) Coated platelets function in platelet-dependent fibrin formation via integrin αIIbβ3 and transglutaminase factor XIII. Haematologica
Mitrugno A, Tormoen GW, Kuhn P, et al. (2015) The prothrombotic activity of cancer cells in the circulation. Blood Reviews
King MR, Phillips KG, Mitrugno A, et al. (2015) A physical sciences network characterization of circulating tumor cell aggregate transport. American Journal of Physiology. Cell Physiology. 308: C792-802
Puy C, Tucker EI, Matafonov A, et al. (2015) Activated factor XI increases the procoagulant activity of the extrinsic pathway by inactivating tissue factor pathway inhibitor. Blood. 125: 1488-96
Loren CP, Aslan JE, Rigg RA, et al. (2015) The BCR-ABL inhibitor ponatinib inhibits platelet immunoreceptor tyrosine-based activation motif (ITAM) signaling, platelet activation and aggregate formation under shear. Thrombosis Research. 135: 155-60
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