Lawrence L. Rudel
Affiliations: | Wake Forest University School of Medicine, Winston-Salem, NC, United States |
Area:
Pathology, BiochemistryWebsite:
https://www.wakehealth.edu/Research/Lipid-Science/Lawrence-L--Rudel,-PhD.htmGoogle:
"Lawrence L. Rudel"Bio:
https://www.bloomberg.com/research/stocks/private/person.asp?personId=321399206&privcapId=204765597&previousCapId=141530835&previousTitle=Cleave%20Biosciences,%20Inc.
https://books.google.com/books?id=RvocAAAAMAAJ
Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Manford Donald Morris | grad student | 1970 | University of Arkansas, Fayetteville | |
| (Studies on exogenous cholesterol metabolism in the normal rabbit, time course of 7α-3H-cholesterol appearance in plasma lipoproteins, liver and intestinal mucosal cells.) | ||||
Children
Sign in to add trainee| Charles W. Joyce | grad student | 2000 | Wake Forest School of Medicine |
| Kirsten J. Williford | grad student | 2000 | Wake Forest School of Medicine |
| Heather M. Alger | grad student | 2009 | Wake Forest School of Medicine |
| Akash Das | grad student | 2009 | Wake Forest School of Medicine |
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Helsley RN, Venkateshwari V, Brown AL, et al. (2019) Obesity-linked suppression of membrane-bound -Acyltransferase 7 (MBOAT7) drives non-alcoholic fatty liver disease. Elife. 8 |
| Ruuth M, Nguyen SD, Vihervaara T, et al. (2018) Susceptibility of low-density lipoprotein particles to aggregate depends on particle lipidome, is modifiable, and associates with future cardiovascular deaths. European Heart Journal |
| Ohshiro T, Kobayashi K, Ohba M, et al. (2017) Selective inhibition of sterolO-acyltransferase 1 isozyme by beauveriolide III in intact cells. Scientific Reports. 7: 4163 |
| Sacks FM, Lichtenstein AH, Wu JHY, et al. (2017) Dietary Fats and Cardiovascular Disease: A Presidential Advisory From the American Heart Association. Circulation |
| Warrier M, Zhang J, Bura K, et al. (2016) Sterol O-Acyltransferase 2-Driven Cholesterol Esterification Opposes Liver X Receptor-Stimulated Fecal Neutral Sterol Loss. Lipids |
| Ohshiro T, Ohtawa M, Nagamitsu T, et al. (2015) New Pyripyropene A Derivatives, Highly SOAT2-Selective Inhibitors, Improve Hypercholesterolemia and Atherosclerosis in Atherogenic Mouse Models. The Journal of Pharmacology and Experimental Therapeutics |
| Lopez AM, Chuang JC, Posey KS, et al. (2015) PRD125, a Potent and Selective Inhibitor of Sterol O-acyltransferase 2 (SOAT2) Markedly Reduces Hepatic Cholesteryl Ester Accumulation and Improves Liver Function in Lysosomal Acid Lipase-deficient Mice. The Journal of Pharmacology and Experimental Therapeutics |
| Melchior JT, Olson JD, Kelley KL, et al. (2015) Targeted Knockdown of Hepatic SOAT2 With Antisense Oligonucleotides Stabilizes Atherosclerotic Plaque in ApoB100-only LDLr-/- Mice. Arteriosclerosis, Thrombosis, and Vascular Biology. 35: 1920-7 |
| Jones PJ, MacKay DS, Senanayake VK, et al. (2015) High-oleic canola oil consumption enriches LDL particle cholesteryl oleate content and reduces LDL proteoglycan binding in humans. Atherosclerosis. 238: 231-8 |
| Shores NJ, Verhague MA, Sawyer JK, et al. (2015) Su1036 Hepatic Apolipoprotein A-IV Gene Expression Is Increased in Non-Alcoholic Fatty Liver Disease and Is Modulated by Gender and Inflammation Gastroenterology. 148: S-1045 |