Lincoln R. Potter
Affiliations: | 1999- | Biochemistry, Molecular Biology and Biophysics | University of Minnesota, Twin Cities, Minneapolis, MN |
Area:
BiochemistryWebsite:
https://cbs.umn.edu/contacts/lincoln-r-potterGoogle:
"Lincoln Ross Potter"Bio:
https://www.scribd.com/doc/54544159/Encyclopedia-of-Biological-Chemistry-Vol-3
Lincoln Potter earned his B.S. in biology/chemistry from David Lipscomb University in Nashville. He received his Ph.D. under the direction of David Garbers from Vanderbilt University in 1994 and then conducted postdoctoral studies with Tony Hunter at the Salk Institute in San Diego. In the fall of 1999, he joined the Department of Biochemistry, Molecular Biology and Biophysics at the University of Minnesota as a tenure-track assistant professor, which is his current position.
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Parents
Sign in to add mentor| David Lorn Garbers | grad student | 1994 | Vanderbilt | |
| (The role of dephosphorylation in the desensitization of guanylyl cylcase-linked natriuretic peptide receptors.) | ||||
| Tony Hunter | post-doc | Salk Institute | ||
Children
Sign in to add trainee| Sarah E. Hosch | grad student | 2004 | UMN |
| Paula M. Bryan | grad student | 2006 | UMN |
| Laura K. Antos | grad student | 2008 | UMN |
| Darcy R. Flora | grad student | 2009 | UMN |
| Andrea R. Yoder | grad student | 2010 | UMN |
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Publications
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| Potter LR. (2024) Phosphorylation-Dependent Regulation of Guanylyl Cyclase (GC)-A and Other Membrane GC Receptors. Endocrine Reviews |
| Andresen H, Pérez-Ternero C, Robinson J, et al. (2023) Novel enhancers of guanylyl cyclase-A activity acting via allosteric modulation. British Journal of Pharmacology |
| Egbert JR, Uliasz TF, Lowther KM, et al. (2022) Epitope-tagged and phosphomimetic mouse models for investigating natriuretic peptide-stimulated receptor guanylyl cyclases. Frontiers in Molecular Neuroscience. 15: 1007026 |
| Robinson JW, Blixt NC, Norton A, et al. (2020) Male mice with elevated C-type natriuretic peptide-dependent guanylyl cyclase-B activity have increased osteoblasts, bone mass and bone strength. Bone. 115320 |
| Edmund AB, Walseth TF, Levinson NM, et al. (2019) The pseudokinase domains of guanylyl cyclase-A and -B allosterically increase the affinity of their catalytic domains for substrate. Science Signaling. 12 |
| Beuve A, Brouckaert P, Burnett, et al. (2019) Receptor guanylyl cyclase (RGC) family (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database Iuphar/Bps Guide to Pharmacology Cite. 2019 |
| Schmidt H, Dickey DM, Dumoulin A, et al. (2018) Regulation of the natriuretic peptide receptor 2 (Npr2) by phosphorylation of juxtamembrane serine and threonine residues is essential for bifurcation of sensory axons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Shuhaibar LC, Robinson JW, Vigone G, et al. (2017) Dephosphorylation of the NPR2 guanylyl cyclase contributes to inhibition of bone growth by fibroblast growth factor. Elife. 6 |
| Robinson JW, Egbert JR, Davydova J, et al. (2017) Dephosphorylation is the mechanism of fibroblast growth factor inhibition of guanylyl cyclase-B. Cellular Signalling |
| Dickey DM, Otto NM, Potter LR. (2017) Skeletal overgrowth-causing mutations mimic an allosterically activated conformation of guanylyl cyclase-B that is inhibited by 2,4,6,-trinitrophenyl ATP. The Journal of Biological Chemistry |