Zoltan Jakus, Dr.
Affiliations: | 2006 | Semmelweis Egyetem, Hungary |
Area:
Immunology, Molecular Biology, PathologyGoogle:
"Zoltan Jakus"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Erzsebet Ligeti | grad student | 2006 | Semmelweis Egyetem | |
| (Integrinek, Fc-receptorok es G-feherje-kapcsolt receptorok jelatvitelenek mechanizmusa neutrofil granulocitakban.) | ||||
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Publications
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| Káposztás E, Balogh L, Mócsai A, et al. (2023) The selective inhibition of the Syk tyrosine kinase ameliorates experimental autoimmune arthritis. Frontiers in Immunology. 14: 1279155 |
| Pawlak JB, Bálint L, Lim L, et al. (2019) Lymphatic mimicry in maternal endothelial cells promotes placental spiral artery remodeling. The Journal of Clinical Investigation |
| Csete D, Simon E, Alatshan A, et al. (2019) Hematopoietic or Osteoclast-Specific Deletion of Syk Leads to Increased Bone Mass in Experimental Mice. Frontiers in Immunology. 10: 937 |
| Németh T, Futosi K, Szabó M, et al. (2019) Importance of Fc Receptor γ-Chain ITAM Tyrosines in Neutrophil Activation and Autoimmune Arthritis. Frontiers in Immunology. 10: 252 |
| Nemeth T, Futosi K, Szabo M, et al. (2018) SAT0002 The intracellular itam tyrosines of fc receptor gamma-chain are critical for experimental autoimmune arthritis Annals of the Rheumatic Diseases |
| Jakus Z. (2016) Fine-tuning of integrin activation. Blood. 127: 275-6 |
| Zhou Z, Rawnsley DR, Goddard LM, et al. (2015) The cerebral cavernous malformation pathway controls cardiac development via regulation of endocardial MEKK3 signaling and KLF expression. Developmental Cell. 32: 168-80 |
| Kovács M, Németh T, Jakus Z, et al. (2015) A2.21 The role of HCK, FGR and LYN inin vivoinflammation in mice Annals of the Rheumatic Diseases. 74: A24.1-A24 |
| Kása O, Futosi K, Trendl J, et al. (2015) A2.12 Phospholipase Cγ2 expression in neutrophils mediates autoantibody-induced arthritis Annals of the Rheumatic Diseases. 74: A20.2-A20 |
| Kovács M, Németh T, Jakus Z, et al. (2014) The Src family kinases Hck, Fgr, and Lyn are critical for the generation of the in vivo inflammatory environment without a direct role in leukocyte recruitment. The Journal of Experimental Medicine. 211: 1993-2011 |