Jeremy T. Copp, Ph.D.
Affiliations: | 2003 | University of California, San Diego, La Jolla, CA |
Area:
Biochemistry, Cell BiologyGoogle:
"Jeremy Copp"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Peter v. der Geer | grad student | 2003 | UCSD | |
| (Normal and pathological activation of receptor protein -tyrosine kinases: A case of molecular mimicry, a taste for salt, and why do we give a Shc?) | ||||
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Publications
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| Oku S, van der Meulen T, Copp J, et al. (2012) Engineering NGF receptors to bind Grb2 directly uncovers differences in signaling ability between Grb2- and ShcA-binding sites. Febs Letters. 586: 3658-64 |
| Copp J, Manning G, Hunter T. (2009) TORC-specific phosphorylation of mammalian target of rapamycin (mTOR): phospho-Ser2481 is a marker for intact mTOR signaling complex 2. Cancer Research. 69: 1821-7 |
| Copp J, Wiley S, Ward MW, et al. (2005) Hypertonic shock inhibits growth factor receptor signaling, induces caspase-3 activation, and causes reversible fragmentation of the mitochondrial network. American Journal of Physiology. Cell Physiology. 288: C403-15 |
| Banerjee M, Copp J, Vuga D, et al. (2004) GW domains of the Listeria monocytogenes invasion protein InlB are required for potentiation of Met activation. Molecular Microbiology. 52: 257-71 |
| Marino M, Banerjee M, Copp J, et al. (2004) Characterization of the calcium-binding sites of Listeria monocytogenes InlB. Biochemical and Biophysical Research Communications. 316: 379-86 |
| Copp J, Marino M, Banerjee M, et al. (2003) Multiple regions of internalin B contribute to its ability to turn on the Ras-mitogen-activated protein kinase pathway. The Journal of Biological Chemistry. 278: 7783-9 |