Hsueh-Ning Liu, Ph.D.

Affiliations: 
2003 McGill University, Montreal, QC, Canada 
Area:
Pharmacology, Biochemistry
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"Hsueh-Ning Liu"
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Guillermina Almazan grad student 2003 McGill
 (Glutamatergic signaling in oligodendrocyte progenitors.)
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Publications

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Hossain S, Liu HN, Fragoso G, et al. (2014) Agonist-induced down-regulation of AMPA receptors in oligodendrocyte progenitors. Neuropharmacology. 79: 506-14
Liu HN, Tjostheim S, Dasilva K, et al. (2012) Targeting of monomer/misfolded SOD1 as a therapeutic strategy for amyotrophic lateral sclerosis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 8791-9
Kerman A, Liu HN, Croul S, et al. (2010) Amyotrophic lateral sclerosis is a non-amyloid disease in which extensive misfolding of SOD1 is unique to the familial form Acta Neuropathologica. 119: 335-344
Silvertown JD, Neschadim A, Liu HN, et al. (2010) Relaxin-3 and receptors in the human and rhesus brain and reproductive tissues. Regulatory Peptides. 159: 44-53
Liu HN, Sanelli T, Horne P, et al. (2009) Lack of evidence of monomer/misfolded superoxide dismutase-1 in sporadic amyotrophic lateral sclerosis. Annals of Neurology. 66: 75-80
Hossain S, Liu HN, Nguyen M, et al. (2009) Cadmium exposure induces mitochondria-dependent apoptosis in oligodendrocytes. Neurotoxicology. 30: 544-54
Larocque D, Galarneau A, Liu HN, et al. (2005) Protection of p27(Kip1) mRNA by quaking RNA binding proteins promotes oligodendrocyte differentiation. Nature Neuroscience. 8: 27-33
Fragoso G, Martínez-Bermúdez AK, Liu HN, et al. (2004) Developmental differences in HO-induced oligodendrocyte cell death: role of glutathione, mitogen-activated protein kinases and caspase 3. Journal of Neurochemistry. 90: 392-404
Brault S, Martinez-Bermudez AK, Roberts J, et al. (2004) Cytotoxicity of the E(2)-isoprostane 15-E(2t)-IsoP on oligodendrocyte progenitors. Free Radical Biology & Medicine. 37: 358-66
Molina-Holgado E, Khorchid A, Liu HN, et al. (2003) Regulation of muscarinic receptor function in developing oligodendrocytes by agonist exposure. British Journal of Pharmacology. 138: 47-56
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