Pin-Lan Li
Affiliations: | Medical College of Wisconsin, Milwaukee, WI, United States |
Area:
Pharmacology, BiochemistryGoogle:
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Publications
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Yuan X, Bhat OM, Lohner H, et al. (2020) Downregulation of Lysosomal Acid Ceramidase Mediates HMGB1-Induced Migration and Proliferation of Mouse Coronary Arterial Myocytes. Frontiers in Cell and Developmental Biology. 8: 111 |
Bhat OM, Yuan X, Camus S, et al. (2020) Abnormal Lysosomal Positioning and Small Extracellular Vesicle Secretion in Arterial Stiffening and Calcification of Mice Lacking Mucolipin 1 Gene. International Journal of Molecular Sciences. 21 |
Li G, Kidd J, Li PL. (2020) Podocyte Lysosome Dysfunction in Chronic Glomerular Diseases. International Journal of Molecular Sciences. 21 |
Bhat OM, Li G, Yuan X, et al. (2020) Arterial Medial Calcification through Enhanced small Extracellular Vesicle Release in Smooth Muscle-Specific Asah1 Gene Knockout Mice. Scientific Reports. 10: 1645 |
Huang D, Li G, Camus S, et al. (2020) Contribution of NLRP3 Inflammasome Activation to Glomerular Injury during Hyperhomocysteinemia with and without Enhanced Exosome Secretion The Faseb Journal. 34: 1-1 |
Bhat OM, Yuan X, Li G, et al. (2020) Regulatory Role of Ceramide/mTOR Signaling in Exosome Secretion from Smooth Muscle Cells during Arterial Stiffening and Medial Calcification The Faseb Journal. 34: 1-1 |
Bhat O, Yuan X, Cain C, et al. (2019) Medial calcification in the arterial wall of smooth muscle cell-specific Smpd1 transgenic mice: A ceramide-mediated vasculopathy. Journal of Cellular and Molecular Medicine |
Hong J, Li G, Zhang Q, et al. (2019) D-Ribose Induces Podocyte NLRP3 Inflammasome Activation and Glomerular Injury via AGEs/RAGE Pathway. Frontiers in Cell and Developmental Biology. 7: 259 |
Zhang Y, Chen Y, Zhang Y, et al. (2019) Contribution of cathepsin B-dependent Nlrp3 inflammasome activation to nicotine-induced endothelial barrier dysfunction. European Journal of Pharmacology. 172795 |
Yuan X, Bhat OM, Lohner H, et al. (2019) Endothelial acid ceramidase in exosome-mediated release of NLRP3 inflammasome products during hyperglycemia: Evidence from endothelium-specific deletion of Asah1 gene. Biochimica Et Biophysica Acta. Molecular and Cell Biology of Lipids. 158532 |