Tsz K. Fung, Ph.D.
Affiliations: | 2006 | Hong Kong University of Science and Technology, Clear Water Bay, Kowloon, Hong Kong |
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Cell BiologyGoogle:
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Parents
Sign in to add mentorRandy Y. C. Poon | grad student | 2006 | HKUST | |
(The functions and proteolysis of cyclin A and cyclin F during mitosis.) |
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Publications
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Luo H, Zhu G, Xu J, et al. (2019) HOTTIP lncRNA Promotes Hematopoietic Stem Cell Self-Renewal Leading to AML-like Disease in Mice. Cancer Cell |
Luo H, Zhu G, Fung TK, et al. (2019) Hottip Lncrna Reinforces CTCF Defined Chromatin Boundaries and Drives Wnt Target Gene Expression in AML Leukemogenesis Blood. 134: 277-277 |
Fung TK, Zeisig B, Zarowiecki M, et al. (2019) Reconstruction of Human AML Reveals Stem Cell Origin and Therapeutic Targets for Treatment Resistant CD34-/Lo MLL-Rearranged Leukemia Blood. 134: 3203-3203 |
Siriboonpiputtana T, Zeisig BB, Zarowiecki M, et al. (2017) Transcriptional memory of cells of origin overrides β-catenin requirement of MLL cancer stem cells. The Embo Journal |
Cheung N, Fung TK, Zeisig BB, et al. (2016) Targeting Aberrant Epigenetic Networks Mediated by PRMT1 and KDM4C in Acute Myeloid Leukemia. Cancer Cell. 29: 32-48 |
Esposito MT, Zhao L, Fung TK, et al. (2015) Synthetic lethal targeting of oncogenic transcription factors in acute leukemia by PARP inhibitors. Nature Medicine |
Man CH, Fung TK, Wan H, et al. (2015) Suppression of SOX7 by DNA methylation and its tumor suppressor function in acute myeloid leukemia. Blood. 125: 3928-36 |
Fung TK, So CWE. (2013) Overcoming treatment resistance in acute promyelocytic leukemia and beyond Oncotarget. 4: 1128-1129 |
Fung TK, Gandillet A, So CW. (2012) Selective treatment of mixed-lineage leukemia leukemic stem cells through targeting glycogen synthase kinase 3 and the canonical Wnt/β-catenin pathway. Current Opinion in Hematology. 19: 280-6 |
Man CH, Fung TK, Ho C, et al. (2012) Sorafenib treatment of FLT3-ITD(+) acute myeloid leukemia: favorable initial outcome and mechanisms of subsequent nonresponsiveness associated with the emergence of a D835 mutation. Blood. 119: 5133-43 |