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Travis I. Moore, Ph.D.

Affiliations: 
2010 Biological Sciences - Ph.D. University of California, Irvine, Irvine, CA 
Area:
Cell Biology
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Tau-Mu Yi grad student 2010 UC Irvine
 (Determining which way to go: How G-proteins and membrane microdomains direct cell polarization and the chemotropic response.)

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Publications

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Moore TI, Aaron J, Chew TL, et al. (2018) Measuring Integrin Conformational Change on the Cell Surface with Super-Resolution Microscopy. Cell Reports. 22: 1903-1912
Nordenfelt P, Moore TI, Mehta SB, et al. (2017) Direction of actin flow dictates integrin LFA-1 orientation during leukocyte migration. Nature Communications. 8: 2047
Swaminathan V, Kalappurakkal JM, Mehta SB, et al. (2017) Actin retrograde flow actively aligns and orients ligand-engaged integrins in focal adhesions. Proceedings of the National Academy of Sciences of the United States of America. 114: 10648-10653
Chou CS, Moore TI, Nie Q, et al. (2015) Alternative cell polarity behaviours arise from changes in G-protein spatial dynamics. Iet Systems Biology. 9: 52-63
Quiñones GA, Moore TI, Nicholes K, et al. (2013) Application of a new wall-less plate technology to complex multistep cell-based investigations using suspension cells. Blood. 121: e25-33
Moore TI, Tanaka H, Kim HJ, et al. (2013) Yeast G-proteins mediate directional sensing and polarization behaviors in response to changes in pheromone gradient direction. Molecular Biology of the Cell. 24: 521-34
Chou CS, Moore TI, Chang SD, et al. (2012) Signaling regulated endocytosis and exocytosis lead to mating pheromone concentration dependent morphologies in yeast. Febs Letters. 586: 4208-14
Moore TI, Chou CS, Nie Q, et al. (2008) Robust spatial sensing of mating pheromone gradients by yeast cells. Plos One. 3: e3865
Waksman R, McEwan PE, Moore TI, et al. (2008) PhotoPoint photodynamic therapy promotes stabilization of atherosclerotic plaques and inhibits plaque progression. Journal of the American College of Cardiology. 52: 1024-32
Deane JA, Kharas MG, Oak JS, et al. (2007) T-cell function is partially maintained in the absence of class IA phosphoinositide 3-kinase signaling. Blood. 109: 2894-902
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