Eugene J. Wyatt, Ph.D.
Affiliations: | 2012 | Integrated Graduate Program in the Life Sciences | Northwestern University, Evanston, IL |
Area:
Cell BiologyGoogle:
"Eugene Wyatt"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Hossein Ardehali | grad student | 2012 | Northwestern | |
| (Hexokinase expression and mitochondrial binding in cytoprotection and the cardiomyocyte response to stress.) | ||||
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Publications
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| Marcheva B, Perelis M, Weidemann BJ, et al. (2020) A role for alternative splicing in circadian control of exocytosis and glucose homeostasis. Genes & Development |
| Demonbreun AR, Wyatt EJ, Fallon KS, et al. (2019) A gene-edited mouse model of Limb-Girdle muscular dystrophy 2C for testing exon skipping. Disease Models & Mechanisms |
| Kim EY, Barefield DY, Vo AH, et al. (2019) Distinct pathological signatures in human cellular models of myotonic dystrophy subtypes. Jci Insight |
| Wyatt EJ, Demonbreun AR, Kim EY, et al. (2018) Efficient exon skipping of SGCG mutations mediated by phosphorodiamidate morpholino oligomers. Jci Insight. 3 |
| McNally EM, Wyatt EJ. (2017) Mutation-Based Therapy for Duchenne Muscular Dystrophy: Antisense Treatment Arrives in the Clinic. Circulation. 136: 979-981 |
| Kim EY, Page P, Dellefave-Castillo LM, et al. (2016) Direct reprogramming of urine-derived cells with inducible MyoD for modeling human muscle disease. Skeletal Muscle. 6: 32 |
| McNally EM, Wyatt EJ. (2016) Welcome to the splice age: antisense oligonucleotide-mediated exon skipping gains wider applicability. The Journal of Clinical Investigation. 126: 1236-8 |
| Gao QQ, Wyatt E, Goldstein JA, et al. (2015) Reengineering a transmembrane protein to treat muscular dystrophy using exon skipping. The Journal of Clinical Investigation |
| Wu R, Wyatt E, Chawla K, et al. (2012) Hexokinase II knockdown results in exaggerated cardiac hypertrophy via increased ROS production. Embo Molecular Medicine. 4: 633-46 |
| Wu R, Smeele KM, Wyatt E, et al. (2011) Reduction in hexokinase II levels results in decreased cardiac function and altered remodeling after ischemia/reperfusion injury. Circulation Research. 108: 60-9 |