Rebecca L. Hutcheson, Ph.D.

Affiliations: 
2013 College of Medicine University of South Alabama, Mobile, AL, United States 
Area:
Biochemistry, Medicine and Surgery, Molecular Biology
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"Rebecca Hutcheson"
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Petra Rocic grad student 2013 University of South Alabama
 (Regulation of vascular smooth muscle phenotype during coronary collateral growth in the metabolic syndrome.)
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Publications

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Soler A, Hunter I, Joseph G, et al. (2018) Corrigendum to "Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation" [J. Mol. Cell. Cardiol. 117 (2018) 88-99]. Journal of Molecular and Cellular Cardiology
Soler A, Hunter I, Joseph G, et al. (2018) Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation. Journal of Molecular and Cellular Cardiology
Joseph G, Soler A, Hutcheson R, et al. (2016) Elevated 20-HETE Impairs Coronary Collateral Growth in Metabolic Syndrome Via Endothelial Dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00561.2016
Diane A, Pierce WD, Kelly SE, et al. (2016) Mechanisms of Comorbidities Associated With the Metabolic Syndrome: Insights from the JCR:LA-cp Corpulent Rat Strain. Frontiers in Nutrition. 3: 44
Hutcheson R, Terry R, Hutcheson B, et al. (2015) miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome. American Journal of Physiology. Heart and Circulatory Physiology. 308: H1323-35
Hutcheson R, Chaplin J, Hutcheson B, et al. (2014) miR-21 normalizes vascular smooth muscle proliferation and improves coronary collateral growth in metabolic syndrome. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 28: 4088-99
Dodd T, Wiggins L, Hutcheson R, et al. (2013) Impaired coronary collateral growth in the metabolic syndrome is in part mediated by matrix metalloproteinase 12-dependent production of endostatin and angiostatin. Arteriosclerosis, Thrombosis, and Vascular Biology. 33: 1339-49
Hutcheson R, Terry R, Chaplin J, et al. (2013) MicroRNA-145 restores contractile vascular smooth muscle phenotype and coronary collateral growth in the metabolic syndrome. Arteriosclerosis, Thrombosis, and Vascular Biology. 33: 727-36
Hutcheson R, Rocic P. (2012) The metabolic syndrome, oxidative stress, environment, and cardiovascular disease: the great exploration. Experimental Diabetes Research. 2012: 271028
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