Pengda Liu, Ph.D.
Affiliations: | 2008 | East Carolina University, Greenville, NC, United States |
Area:
Molecular Biology, Cell BiologyGoogle:
"Pengda Liu"Mean distance: (not calculated yet)
Parents
Sign in to add mentorJohn W. Stiller | grad student | 2008 | East Carolina University | |
(Catching the wagging tail: Decoding the carboxil terminal domain of RNAP II Rpb1 in Saccharomyces cerevisiae.) |
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Publications
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Antonia RJ, Castillo J, Herring LE, et al. (2019) TBK1 Limits mTORC1 by Promoting Phosphorylation of Raptor Ser877. Scientific Reports. 9: 13470 |
Jiang Y, Zhang Y, Leung JY, et al. (2019) MERTK mediated novel site Akt phosphorylation alleviates SAV1 suppression. Nature Communications. 10: 1515 |
Liu P, Gan W, Inuzuka H, et al. (2019) Author Correction: Sin1 phosphorylation impairs mTORC2 complex integrity and inhibits downstream Akt signalling to suppress tumorigenesis. Nature Cell Biology |
Dai X, Gan W, Li X, et al. (2017) Prostate cancer-associated SPOP mutations confer resistance to BET inhibitors through stabilization of BRD4. Nature Medicine |
Wang B, Jie Z, Joo D, et al. (2017) TRAF2 and OTUD7B govern a ubiquitin-dependent switch that regulates mTORC2 signalling. Nature |
Wan L, Chen M, Cao J, et al. (2017) The APC/C E3 Ligase Complex Activator FZR1 Restricts BRAF Oncogenic Function. Cancer Discovery |
Guo J, Chakraborty AA, Liu P, et al. (2016) pVHL suppresses kinase activity of Akt in a proline-hydroxylation-dependent manner. Science (New York, N.Y.). 353: 929-32 |
Zhang J, Xu K, Liu P, et al. (2016) Inhibition of Rb Phosphorylation Leads to mTORC2-Mediated Activation of Akt. Molecular Cell |
Kim BM, Hong Y, Lee S, et al. (2015) Therapeutic Implications for Overcoming Radiation Resistance in Cancer Therapy. International Journal of Molecular Sciences. 16: 26880-26913 |
Gan W, Dai X, Lunardi A, et al. (2015) SPOP Promotes Ubiquitination and Degradation of the ERG Oncoprotein to Suppress Prostate Cancer Progression. Molecular Cell |