David Dweck, Ph.D.
Affiliations: | 2008 | University of Miami, Coral Gables, FL |
Area:
Pharmacology, Biochemistry, Molecular BiologyGoogle:
"David Dweck"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| James D. Potter | grad student | 2008 | University of Miami | |
| (Challenging current paradigms related to cardiomyopathies: Are changes in the calcium sensitivity of myofilaments containing mutations good predictors of the phenotypic outcomes?) | ||||
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| Veltri T, Landim-Vieira M, Parvatiyar MS, et al. (2017) Hypertrophic Cardiomyopathy Cardiac Troponin C Mutations Differentially Affect Slow Skeletal and Cardiac Muscle Regulation. Frontiers in Physiology. 8: 221 |
| Vukmirovic M, Sanchez-Gonzalez MA, Frye GS, et al. (2015) Structural and Functional Changes in Skeletal Muscles in an A8V-Troponin C Hypertrophic Cardiomyopathy Knock-In Mouse Model Biophysical Journal. 108: 594a |
| Veltri T, Michell CA, Landim M, et al. (2015) The Functional Consequences of Hypertrophic Cardiomyopathy Troponin C Mutations in the Regulation of Slow Skeletal Muscle Contraction: The Protective Role of Slow Skeletal Troponin I Biophysical Journal. 108: 593a-594a |
| Dweck D, Sanchez-Gonzalez MA, Chang AN, et al. (2014) Long term ablation of protein kinase A (PKA)-mediated cardiac troponin I phosphorylation leads to excitation-contraction uncoupling and diastolic dysfunction in a knock-in mouse model of hypertrophic cardiomyopathy. The Journal of Biological Chemistry. 289: 23097-111 |
| Vukmirovic M, Sanchez-Gonzalez MA, Ruiz E, et al. (2014) Myosin Heavy Chain Isoform Switching in Skeletal Muscles in an A8V-Troponin C Hypertrophic Cardiomyopathy Knock-In Mouse Model Biophysical Journal. 106: 775a |
| Martins AS, Parvatiyar MS, Turna R, et al. (2014) In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 is a Hypertrophic Cardiomyopathy Susceptibility Gene Biophysical Journal. 106: 723a |
| Turna RS, Weller RS, Hwang H, et al. (2014) Crossing Mice Carrying TnT Disease Mutations with Opposite Effects on the Myofilament Calcium Sensitivity Partially Normalizes Myofilament Function and Ameliorates Cardiomyopathy Phenotypes Biophysical Journal. 106: 345a |
| Wang Y, Pinto JR, Solis RS, et al. (2012) Generation and functional characterization of knock-in mice harboring the cardiac troponin I-R21C mutation associated with hypertrophic cardiomyopathy. The Journal of Biological Chemistry. 287: 2156-67 |
| Dweck D, Liang J, Potter JD, et al. (2012) The Functional Consequences of HCM Troponin C Mutations in the Regulation of Slow Skeletal Muscle Contraction Biophysical Journal. 102: 155a |
| Pinto JR, Reynaldo DP, Parvatiyar MS, et al. (2011) Strong cross-bridges potentiate the Ca(2+) affinity changes produced by hypertrophic cardiomyopathy cardiac troponin C mutants in myofilaments: a fast kinetic approach. The Journal of Biological Chemistry. 286: 1005-13 |