Lee Chao
Affiliations: | Medical University of South Carolina, Charleston, SC, United States |
Area:
Biochemistry, Molecular BiologyGoogle:
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Children
Sign in to add traineeEric G. Dobrzynski | grad student | 2001 | MUSC |
David A. Montanari | grad student | 2005 | MUSC |
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Publications
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Chao J, Guo Y, Chao L. (2018) Protective Role of Endogenous Kallistatin in Vascular Injury and Senescence by Inhibiting Oxidative Stress and Inflammation. Oxidative Medicine and Cellular Longevity. 2018: 4138560 |
Yao Y, Li B, Liu C, et al. (2018) Reduced Plasma Kallistatin Is Associated With the Severity of Coronary Artery Disease, and Kallistatin Treatment Attenuates Atherosclerotic Plaque Formation in Mice. Journal of the American Heart Association. 7: e009562 |
Chao J, Guo Y, Li P, et al. (2017) Opposing Effects of Oxygen Regulation on Kallistatin Expression: Kallistatin as a Novel Mediator of Oxygen-Induced HIF-1-eNOS-NO Pathway. Oxidative Medicine and Cellular Longevity. 2017: 5262958 |
Guo Y, Li P, Gao L, et al. (2017) Kallistatin reduces vascular senescence and aging by regulating microRNA-34a-SIRT1 pathway. Aging Cell |
Chao J, Bledsoe G, Chao L. (2016) Protective Role of Kallistatin in Vascular and Organ Injury. Hypertension |
Lin WC, Chen CW, Huang YW, et al. (2015) Kallistatin protects against sepsis-related acute lung injury via inhibiting inflammation and apoptosis. Scientific Reports. 5: 12463 |
Chao J, Bledsoe G, Chao L. (2014) Kallikrein-kinin in stem cell therapy. World Journal of Stem Cells. 6: 448-57 |
Chao J, Bledsoe G, Chao L. (2014) Tissue kallikrein-kinin therapy in hypertension and organ damage. Progress in Drug Research. Fortschritte Der Arzneimittelforschung. Progrã¨S Des Recherches Pharmaceutiques. 69: 37-57 |
Li P, Bledsoe G, Yang ZR, et al. (2014) Human kallistatin administration reduces organ injury and improves survival in a mouse model of polymicrobial sepsis. Immunology. 142: 216-26 |
Yao Y, Sheng Z, Li Y, et al. (2013) Tissue kallikrein-modified human endothelial progenitor cell implantation improves cardiac function via enhanced activation of akt and increased angiogenesis. Laboratory Investigation; a Journal of Technical Methods and Pathology. 93: 577-91 |