Michael J. Katovich

Affiliations: 
University of Florida, Gainesville, Gainesville, FL, United States 
Area:
Pharmacology, Molecular Biology, Pharmaceutical Chemistry
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"Michael Katovich"
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Publications

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Sharma RK, Oliveira AC, Kim S, et al. (2018) Involvement of Neuroinflammation in the Pathogenesis of Monocrotaline-Induced Pulmonary Hypertension. Hypertension (Dallas, Tex. : 1979)
Rathinasabapathy A, Horowitz A, Horton K, et al. (2018) The Selective Angiotensin II Type 2 Receptor Agonist, Compound 21, Attenuates the Progression of Lung Fibrosis and Pulmonary Hypertension in an Experimental Model of Bleomycin-Induced Lung Injury. Frontiers in Physiology. 9: 180
Wang LA, de Kloet AD, Smeltzer MD, et al. (2018) Coupling corticotropin-releasing-hormone and angiotensin converting enzyme 2 dampens stress responsiveness in male mice. Neuropharmacology
Cole-Jeffrey CT, Pepine CJ, Katovich MJ, et al. (2017) Beneficial Effects Of Angiotensin-(1-7) On Cd34 + Cells From Heart Failure Patients. Journal of Cardiovascular Pharmacology
Rathinasabapathy A, Bruce E, Espejo A, et al. (2016) Therapeutic potential of adipose stem cells derived conditioned medium against pulmonary hypertension and lung fibrosis. British Journal of Pharmacology
Wang L, de Kloet AD, Pati D, et al. (2016) Increasing brain angiotensin converting enzyme 2 activity decreases anxiety-like behavior in male mice by activating central Mas receptors. Neuropharmacology
Pei N, Wan R, Chen X, et al. (2015) Angiotensin-(1-7) Decreases Cell Growth and Angiogenesis of Human Nasopharyngeal Carcinoma Xenografts. Molecular Cancer Therapeutics
Qi YF, Zhang J, Wang L, et al. (2015) Angiotensin-converting enzyme 2 inhibits high-mobility group box 1 and attenuates cardiac dysfunction post-myocardial ischemia. Journal of Molecular Medicine (Berlin, Germany)
Cole-Jeffrey CT, Liu M, Katovich MJ, et al. (2015) ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy. Journal of Cardiovascular Pharmacology
Bruce E, Shenoy V, Rathinasabapathy A, et al. (2015) Selective activation of angiotensin AT2 receptors attenuates progression of pulmonary hypertension and inhibits cardiopulmonary fibrosis. British Journal of Pharmacology. 172: 2219-31
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