Wylie Vale
Affiliations: | Salk Institute for Biological Studies, La Jolla, CA, United States |
Area:
NeuroendocrinologyGoogle:
"Wylie Vale"Mean distance: 14.08 (cluster 6) | S | N | B | C | P |
Children
Sign in to add trainee| Christopher P. Horoszko | research assistant | Salk Institute (Chemistry Tree) | |
| Hannah Park | research assistant | 2006-2009 | Salk Institute |
| Ezra M. Wiater | grad student | 2003 | UCSD |
| Brook L. Henry | grad student | 2006 | UCSD |
| Jonathan A. kelber | grad student | 2004-2009 | UCSD |
| Tracy Bale | post-doc | Salk Institute | |
| Alon Chen | post-doc | Salk Institute | |
| David A. Lovejoy | post-doc | Salk Institute | |
| Robert M. Sapolsky | post-doc | Salk Institute | |
| Mary Stenzel-Poore | post-doc | Salk Institute | |
| Elizabeth I. Flandreau | post-doc | 2009-2012 | Salk Institute |
| Joachim Spiess | research scientist |
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Dautzenberg FM, Grigoriadis DE, Hauger RL, et al. (2019) Corticotropin-releasing factor receptors (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database Iuphar/Bps Guide to Pharmacology Cite. 2019 |
| Flandreau EI, Bourke CH, Ressler KJ, et al. (2013) Escitalopram alters gene expression and HPA axis reactivity in rats following chronic overexpression of corticotropin-releasing factor from the central amygdala. Psychoneuroendocrinology. 38: 1349-61 |
| Neufeld-Cohen A, Kelly PA, Paul ED, et al. (2012) Chronic activation of corticotropin-releasing factor type 2 receptors reveals a key role for 5-HT1A receptor responsiveness in mediating behavioral and serotonergic responses to stressful challenge. Biological Psychiatry. 72: 437-47 |
| Wiater E, Vale W. (2012) Roles of activin family in pancreatic development and homeostasis. Molecular and Cellular Endocrinology. 359: 23-9 |
| Bilezikjian LM, Justice NJ, Blackler AN, et al. (2012) Cell-type specific modulation of pituitary cells by activin, inhibin and follistatin. Molecular and Cellular Endocrinology. 359: 43-52 |
| Spike BT, Kelber JA, Kalathur M, et al. (2012) Abstract 3493: Soluble ALK4 extracellular domain antagonizes the cancer/stem cell phenotype caused by Nodal and Cripto signaling in mammary epithelia Cancer Research. 72: 3493-3493 |
| Bilezikjian LM, Vale WW. (2011) The Local Control of the Pituitary by Activin Signaling and Modulation. Open Neuroendocrinology Journal (Online). 4: 90-101 |
| Justice NJ, Blount AL, Pelosi E, et al. (2011) Impaired FSHbeta expression in the pituitaries of Foxl2 mutant animals. Molecular Endocrinology (Baltimore, Md.). 25: 1404-15 |
| Jamieson PM, Cleasby ME, Kuperman Y, et al. (2011) Urocortin 3 transgenic mice exhibit a metabolically favourable phenotype resisting obesity and hyperglycaemia on a high-fat diet. Diabetologia. 54: 2392-403 |
| Fekete EM, Zhao Y, Szücs A, et al. (2011) Systemic urocortin 2, but not urocortin 1 or stressin 1-A, suppresses feeding via CRF2 receptors without malaise and stress. British Journal of Pharmacology. 164: 1959-75 |