Matthew D. Bruss, Ph.D.
Affiliations: | 2011 | Molecular & Biochemical Nutrition | University of California, Berkeley, Berkeley, CA, United States |
Area:
Nutrition, Physiology Biology, Cell BiologyGoogle:
"Matthew Bruss"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Marc K. Hellerstein | grad student | 2011 | UC Berkeley | |
| (Effects of Physiologic, Metabolic and Molecular Adaptations to Calorie Restriction on Biomarkers of Longevity.) | ||||
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| Thompson AC, Bruss MD, Price JC, et al. (2015) Reduced in vivo hepatic proteome replacement rates but not cell proliferation rates predict maximum lifespan extension in mice. Aging Cell |
| Thompson AC, Bruss MD, Nag N, et al. (2014) Fibroblast growth factor 21 is not required for the reductions in circulating insulin-like growth factor-1 or global cell proliferation rates in response to moderate calorie restriction in adult mice. Plos One. 9: e111418 |
| Miller BF, Robinson MM, Reuland DJ, et al. (2013) Calorie restriction does not increase short-term or long-term protein synthesis. The Journals of Gerontology. Series a, Biological Sciences and Medical Sciences. 68: 530-8 |
| Price JC, Khambatta CF, Li KW, et al. (2012) The effect of long term calorie restriction on in vivo hepatic proteostatis: a novel combination of dynamic and quantitative proteomics. Molecular & Cellular Proteomics : McP. 11: 1801-14 |
| Miller BF, Robinson MM, Bruss MD, et al. (2012) A comprehensive assessment of mitochondrial protein synthesis and cellular proliferation with age and caloric restriction. Aging Cell. 11: 150-61 |
| Ahmadian M, Abbott MJ, Tang T, et al. (2011) Desnutrin/ATGL is regulated by AMPK and is required for a brown adipose phenotype. Cell Metabolism. 13: 739-48 |
| Bruss MD, Thompson AC, Aggarwal I, et al. (2011) The effects of physiological adaptations to calorie restriction on global cell proliferation rates. American Journal of Physiology. Endocrinology and Metabolism. 300: E735-45 |
| Bruss MD, Khambatta CF, Ruby MA, et al. (2010) Calorie restriction increases fatty acid synthesis and whole body fat oxidation rates. American Journal of Physiology. Endocrinology and Metabolism. 298: E108-16 |
| Bruss MD, Arias EB, Lienhard GE, et al. (2005) Increased phosphorylation of Akt substrate of 160 kDa (AS160) in rat skeletal muscle in response to insulin or contractile activity. Diabetes. 54: 41-50 |