Shan Zha, Ph.D.

1998-2004 Pharmacology and Molecular Sciences Johns Hopkins University, Baltimore, MD 
 2004-2009 Molecular Genetics Harvard - Boston Children's Hospital 
 2009- Institute for Cancer Genetics Columbia University College of Physicians and Surgeons, New York, NY, United States 
DNA repair, Oncology, Molecular Biology, Pharmacology
"Shan Zha"
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William B. Isaacs grad student 1998-2004 Johns Hopkins
 (Cyclooxygenase-2 and alpha-methylacyl-CoA racemase in prostate cancer.)
Frederick W. Alt post-doc 2004-2009 Harvard - Boston Children's Hospital
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Liu X, Liu T, Shang Y, et al. (2020) ERCC6L2 promotes DNA orientation-specific recombination in mammalian cells. Cell Research
Tao Y, Li M, Liu X, et al. (2020) Dual-color plasmonic nanosensor for radiation dosimetry. Acs Applied Materials & Interfaces
Shao Z, Flynn RA, Crowe JL, et al. (2020) DNA-PKcs has KU-dependent function in rRNA processing and haematopoiesis. Nature
Jiang W, Estes VM, Wang XS, et al. (2019) Phosphorylation at S2053 in Murine (S2056 in Human) DNA-PKcs Is Dispensable for Lymphocyte Development and Class Switch Recombination. Journal of Immunology (Baltimore, Md. : 1950)
Liu X, Wang XS, Lee BJ, et al. (2019) CtIP is essential for early B cell proliferation and development in mice. The Journal of Experimental Medicine
Menolfi D, Jiang W, Lee BJ, et al. (2018) Kinase-dead ATR differs from ATR loss by limiting the dynamic exchange of ATR and RPA. Nature Communications. 9: 5351
Billing D, Horiguchi M, Wu-Baer F, et al. (2018) The BRCT Domains of the BRCA1 and BARD1 Tumor Suppressors Differentially Regulate Homology-Directed Repair and Stalled Fork Protection. Molecular Cell
Crowe JL, Shao Z, Wang XS, et al. (2018) Kinase-dependent structural role of DNA-PKcs during immunoglobulin class switch recombination. Proceedings of the National Academy of Sciences of the United States of America
Hung PJ, Johnson B, Chen BR, et al. (2018) MRI Is a DNA Damage Response Adaptor during Classical Non-homologous End Joining. Molecular Cell
Liu X, Shao Z, Jiang W, et al. (2017) PAXX promotes KU accumulation at DNA breaks and is essential for end-joining in XLF-deficient mice. Nature Communications. 8: 13816
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