Jennifer E. Skeen, Ph.D.
Affiliations: | 2006 | University of Illinois at Chicago, Health Sciences Center, Chicago, IL 60612, United States |
Area:
Molecular Biology, Cell BiologyGoogle:
"Jennifer Skeen"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Nissim Hay | grad student | 2006 | University of Illinois at Chicago, Health Sciences Center | |
| (Molecular mechanisms of Akt mediated cellular proliferation and oncogenic transformation.) | ||||
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Publications
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| Skeen JE, Bhaskar PT, Chen CC, et al. (2006) Akt deficiency impairs normal cell proliferation and suppresses oncogenesis in a p53-independent and mTORC1-dependent manner. Cancer Cell. 10: 269-80 |
| Hahn-Windgassen A, Nogueira V, Chen CC, et al. (2005) Akt activates the mammalian target of rapamycin by regulating cellular ATP level and AMPK activity. The Journal of Biological Chemistry. 280: 32081-9 |
| Majewski N, Nogueira V, Bhaskar P, et al. (2004) Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak. Molecular Cell. 16: 819-30 |
| King FW, Skeen J, Hay N, et al. (2004) Inhibition of Chk1 by activated PKB/Akt. Cell Cycle (Georgetown, Tex.). 3: 634-7 |
| Peng XD, Xu PZ, Chen ML, et al. (2003) Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2. Genes & Development. 17: 1352-65 |
| Kandel ES, Skeen J, Majewski N, et al. (2002) Activation of Akt/protein kinase B overcomes a G(2)/m cell cycle checkpoint induced by DNA damage. Molecular and Cellular Biology. 22: 7831-41 |