Charles J. Bieberich

Affiliations: 
Biological Sciences University of Maryland, Baltimore County 
Area:
Oncology, Biochemistry, Molecular Biology
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"Charles Bieberich"
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Publications

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Hao L, Thomas S, Greer T, et al. (2019) Quantitative proteomic analysis of a genetically induced prostate inflammation mouse model via custom 4-plex DiLeu isobaric labeling. American Journal of Physiology. Renal Physiology
Ashok A, Keener RA, Rubenstein M, et al. (2019) Consequences of interleukin 1β-triggered chronic inflammation in the mouse prostate gland: Altered architecture associated with prolonged CD4 infiltration mimics human proliferative inflammatory atrophy. The Prostate
Sfanos KS, Yegnasubramanian S, Nelson WG, et al. (2019) If this is true, what does it imply? How end-user antibody validation facilitates insights into biology and disease. Asian Journal of Urology. 6: 10-25
Xu X, Wu J, Liu S, et al. (2018) Redox-Responsive Nanoparticle-Mediated Systemic RNAi for Effective Cancer Therapy. Small (Weinheim An Der Bergstrasse, Germany). e1802565
Markowski MC, Hubbard GK, Hicks JL, et al. (2018) Characterization of novel cell lines derived from a MYC-driven murine model of lethal metastatic adenocarcinoma of the prostate. The Prostate
Sreenath TL, Macalindong SS, Mikhalkevich N, et al. (2017) Erratum: ETS Related Gene mediated Androgen Receptor Aggregation and Endoplasmic Reticulum Stress in Prostate Cancer Development. Scientific Reports. 7: 10693
Sreenath TL, Macalindong SS, Mikhalkevich N, et al. (2017) ETS Related Gene mediated Androgen Receptor Aggregation and Endoplasmic Reticulum Stress in Prostate Cancer Development. Scientific Reports. 7: 1109
Li X, Cox JT, Huang W, et al. (2016) Quantifying Kinase-specific Phosphorylation Stoichiometry Using Stable Isotope Labeling In a Reverse In-gel Kinase Assay. Analytical Chemistry
Padmanabhan A, Rao V, De Marzo AM, et al. (2016) Regulating NKX3.1 stability and function: Post-translational modifications and structural determinants. The Prostate. 76: 523-33
Hubbard GK, Mutton LN, Khalili M, et al. (2015) Combined MYC activation and Pten loss are sufficient to create genomic instability and lethal metastatic prostate cancer. Cancer Research
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