Scott Allen Armstrong
Affiliations: | Biology, Molecular and Cellular | Harvard University, Cambridge, MA, United States |
Area:
General Biology, Molecular Biology, Biochemistry, OncologyWebsite:
https://hsci.harvard.edu/people/scott-armstrong-md-phdGoogle:
"Scott Allen Armstrong"Bio:
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Parents
Sign in to add mentor| Michael Stuart Brown | grad student | 1994 | UT Southwestern | |
| (Molecular and biochemical characterization of Rab geranylgeranyl transferase.) | ||||
| Joseph L. Goldstein | grad student | 1994 | UT Southwestern | |
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Publications
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| Heikamp EB, Martucci C, Henrich JA, et al. (2024) NUP98 fusion proteins and KMT2A-MENIN antagonize PRC1.1 to drive gene expression in AML. Cell Reports. 43: 114901 |
| Kwon MC, Thuring JW, Querolle O, et al. (2024) Preclinical efficacy of potent and selective menin-KMT2A inhibitor JNJ-75276617 in KMT2A- and NPM1-altered leukemias. Blood |
| Smolko AE, Sullivan DW, Olsen SN, et al. (2024) A MOZ-TIF2 leukemia mouse model displays KAT6-dependent H3K23 propionylation and overexpression of a set of active developmental genes. Proceedings of the National Academy of Sciences of the United States of America. 121: e2405905121 |
| Gu H, Chen C, Hou ZS, et al. (2024) PI3Kγ maintains the self-renewal of acute myeloid leukemia stem cells by regulating the pentose phosphate pathway. Blood |
| Tubío-Santamaría N, Jayavelu AK, Schnoeder TM, et al. (2023) Immunoproteasome function maintains oncogenic gene expression in KMT2A-complex driven leukemia. Molecular Cancer. 22: 196 |
| Issa GC, Aldoss I, DiPersio J, et al. (2023) The menin inhibitor revumenib in KMT2A-rearranged or NPM1-mutant leukaemia. Nature |
| Uckelmann HJ, Haarer EL, Takeda R, et al. (2022) Mutant NPM1 directly regulates oncogenic transcription in acute myeloid leukemia. Cancer Discovery |
| Soto-Feliciano YM, Sanchez-Rivera FJ, Perner F, et al. (2022) A molecular switch between mammalian MLL complexes dictates response to Menin-MLL inhibition. Cancer Discovery |
| Aubrey BJ, Cutler JA, Bourgeois W, et al. (2022) IKAROS and MENIN coordinate therapeutically actionable leukemogenic gene expression in MLL-r acute myeloid leukemia. Nature Cancer |
| Heikamp EB, Henrich JA, Perner F, et al. (2021) The Menin-MLL1 interaction is a molecular dependency in NUP98-rearranged AML. Blood |