Svetlana Lutsenko
Affiliations: | Physiology | Johns Hopkins University, Baltimore, MD |
Area:
Physiology Biology, Biochemistry, Cell BiologyGoogle:
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Publications
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| Kabin E, Dong Y, Roy S, et al. (2023) α-lipoic acid ameliorates consequences of copper overload by up-regulating selenoproteins and decreasing redox misbalance. Proceedings of the National Academy of Sciences of the United States of America. 120: e2305961120 |
| Washington-Hughes CL, Roy S, Seneviratne HK, et al. (2023) Atp7b-dependent choroid plexus dysfunction Causes transient copper deficit and metabolic changes in the developing mouse brain. Plos Genetics. 19: e1010558 |
| McCann CJ, Hasan NM, Padilla-Benavides T, et al. (2022) Heterogeneous nuclear ribonucleoprotein hnRNPA2/B1 regulates the abundance of the copper-transporter ATP7A in an isoform-dependent manner. Frontiers in Molecular Biosciences. 9: 1067490 |
| Dev S, Muchenditsi A, Gottlieb A, et al. (2022) Oxysterol misbalance critically contributes to Wilson disease pathogenesis. Science Advances. 8: eadc9022 |
| Dev S, Kruse RL, Hamilton JP, et al. (2022) Wilson Disease: Update on Pathophysiology and Treatment. Frontiers in Cell and Developmental Biology. 10: 871877 |
| Tsvetkov P, Coy S, Petrova B, et al. (2022) Copper induces cell death by targeting lipoylated TCA cycle proteins. Science (New York, N.Y.). 375: 1254-1261 |
| Ge EJ, Bush AI, Casini A, et al. (2021) Connecting copper and cancer: from transition metal signalling to metalloplasia. Nature Reviews. Cancer |
| Lutsenko S. (2021) Dynamic and cell-specific transport networks for intracellular copper ions. Journal of Cell Science. 134 |
| Muchenditsi A, Talbot CC, Gottlieb A, et al. (2021) Systemic deletion of Atp7b modifies the hepatocytes' response to copper overload in the mouse models of Wilson disease. Scientific Reports. 11: 5659 |
| Uhlemann EE, Yu CH, Patry J, et al. (2020) Nanobodies against the metal binding domains of ATP7B as tools to study copper transport in the cell. Metallomics : Integrated Biometal Science |