Ronald P. Wong
Affiliations: | Institute of Molecular Biology Mainz, Germany |
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| Renz C, Asimaki E, Meister C, et al. (2023) Ubiquiton-An inducible, linkage-specific polyubiquitylation tool. Molecular Cell |
| Yakoub G, Choi YS, Wong RP, et al. (2023) Avidity-based biosensors for ubiquitylated PCNA reveal choreography of DNA damage bypass. Science Advances. 9: eadf3041 |
| Cabello-Lobato MJ, González-Garrido C, Cano-Linares MI, et al. (2021) Physical interactions between MCM and Rad51 facilitate replication fork lesion bypass and ssDNA gap filling by non-recombinogenic functions. Cell Reports. 36: 109440 |
| Wong RP, Petriukov K, Ulrich HD. (2021) Daughter-strand gaps in DNA replication - substrates of lesion processing and initiators of distress signalling. Dna Repair. 105: 103163 |
| Wong RP, García-Rodríguez N, Zilio N, et al. (2019) Processing of DNA Polymerase-Blocking Lesions during Genome Replication Is Spatially and Temporally Segregated from Replication Forks. Molecular Cell |
| García-Rodríguez N, Wong RP, Ulrich HD. (2018) The helicase Pif1 functions in the template switching pathway of DNA damage bypass. Nucleic Acids Research |
| García-Rodríguez N, Morawska M, Wong RP, et al. (2018) Spatial separation between replisome- and template-induced replication stress signaling. The Embo Journal |
| Hung SH, Wong RP, Ulrich HD, et al. (2017) Monoubiquitylation of histone H2B contributes to the bypass of DNA damage during and after DNA replication. Proceedings of the National Academy of Sciences of the United States of America |
| García-Rodríguez N, Wong RP, Ulrich HD. (2016) Functions of Ubiquitin and SUMO in DNA Replication and Replication Stress. Frontiers in Genetics. 7: 87 |
| Wong RP, Lin H, Khosravi S, et al. (2011) Tumour suppressor ING1b maintains genomic stability upon replication stress. Nucleic Acids Research. 39: 3632-42 |