Gordon F. Tomaselli
Affiliations: | Medicine | Johns Hopkins Medical School/HHMI, Baltimore, MD, United States |
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Collaborators
Sign in to add collaborator| Sandra B. Gabelli | collaborator | 2007- | Johns Hopkins Medical School/HHMI |
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Publications
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| Tomaselli GF. (2023) BIOLOGICAL ANTIARRHYTHMICS-SODIUM CHANNEL INTERACTING PROTEINS. Transactions of the American Clinical and Climatological Association. 133: 136-148 |
| Chakouri N, Rivas S, Roybal D, et al. (2022) Fibroblast growth factor homologous factors serve as a molecular rheostat in tuning arrhythmogenic cardiac late sodium current. Nature Cardiovascular Research. 1: 1-13 |
| Srinivasan L, Alzogaray V, Selvakumar D, et al. (2022) Development of High affinity Nanobodies Specific for Na1.4 and Na1.5 Voltage-Gated Sodium Channel Isoforms. The Journal of Biological Chemistry. 101763 |
| Kang PW, Chakouri N, Diaz J, et al. (2021) Elementary mechanisms of calmodulin regulation of Na1.5 producing divergent arrhythmogenic phenotypes. Proceedings of the National Academy of Sciences of the United States of America. 118 |
| Nathan S, Gabelli SB, Yoder JB, et al. (2021) Structural basis of cytoplasmic NaV1.5 and NaV1.4 regulation. The Journal of General Physiology. 153 |
| McNally EM, Mann DL, Pinto Y, et al. (2020) Clinical Care Recommendations for Cardiologists Treating Adults With Myotonic Dystrophy. Journal of the American Heart Association. 9: e014006 |
| Chakouri N, wei Kang P, Diaz J, et al. (2020) Aberrant Calmodulin Regulation of NaV1.5 Channels Linked to Inherited Cardiac Arrhythmia Biophysical Journal. 118: 577a |
| Yoder JB, Ben-Johny M, Farinelli F, et al. (2019) Ca-dependent regulation of sodium channels Na1.4 and Na1.5 is controlled by the post-IQ motif. Nature Communications. 10: 1514 |
| Nathan S, Shoemaker S, Farinelli F, et al. (2019) An Attempted Molecular Rescue of an Arrhythmogenic Cardiac Disease Mutation Biophysical Journal. 116: 387a |
| Blazeski A, Lowenthal J, Wang Y, et al. (2018) Engineered Heart Slice Model of Arrhythmogenic Cardiomyopathy using Plakophilin-2 Mutant Myocytes. Tissue Engineering. Part A |