Mark Borris Aldonza
Affiliations: | Department of Chemical and Biomolecular Engineering | Korea Advanced Institute of Science and Technology, Daejeon, South Korea |
Area:
cancer biology, resistance evolution, drug resistance, metastasis, systems pharmacology, chemical biologyGoogle:
"Mark Borris Aldonza"Mean distance: (not calculated yet)
Parents
Sign in to add mentorSang Kook Lee | research assistant | 2013-2014 | Seoul National University |
Je-Yoel Cho | research assistant | 2014-2015 | Seoul National University |
Daniel Gerlich | research assistant | 2017-2017 | Institute of Molecular Biotechnology (IMBA) |
Yoosik Kim | research assistant | 2016-2019 | Korea Advanced Inst. of Science and Technology (KAIST) |
Je-Yoel Cho | grad student | 2020-2024 | Seoul National University |
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Publications
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Son KH, Aldonza MBD, Nam AR, et al. (2023) Integrative mapping of the dog epigenome: Reference annotation for comparative intertissue and cross-species studies. Science Advances. 9: eade3399 |
Aldonza MBD, Cha J, Yong I, et al. (2023) Multi-targeted therapy resistance via drug-induced secretome fucosylation. Elife. 12 |
Shin SB, Kim DH, Kim DE, et al. (2021) Dual Targeting of EGFR with PLK1 Exerts Therapeutic Synergism in Taxane-Resistant Lung Adenocarcinoma by Suppressing ABC Transporters. Cancers. 13 |
Aldonza MBD, Reyes RDD, Kim YS, et al. (2021) Chemotherapy confers a conserved secondary tolerance to EGFR inhibition via AXL-mediated signaling bypass Scientific Reports. 11: 8016 |
Aldonza MBD, Reyes RDD, Kim YS, et al. (2021) Chemotherapy confers a conserved secondary tolerance to EGFR inhibition via AXL-mediated signaling bypass. Scientific Reports. 11: 8016 |
Aldonza MB, Cha J, Yong I, et al. (2021) Widespread multi-targeted therapy resistance via drug-induced secretome fucosylation Biorxiv |
Aldonza MBD, Ku J, Hong JY, et al. (2020) Prior acquired resistance to paclitaxel relays diverse EGFR-targeted therapy persistence mechanisms. Science Advances. 6: eaav7416 |
Aldonza MBD, Son YS, Sung HJ, et al. (2017) Paraoxonase-1 (PON1) induces metastatic potential and apoptosis escape via its antioxidative function in lung cancer cells. Oncotarget |
Aldonza MB, Hong JY, Lee SK. (2017) Paclitaxel-resistant cancer cell-derived secretomes elicit ABCB1-associated docetaxel cross-resistance and escape from apoptosis through FOXO3a-driven glycolytic regulation. Experimental & Molecular Medicine. 49: e286 |
Aldonza MB, Hong JY, Alinsug MV, et al. (2016) Multiplicity of acquired cross-resistance in paclitaxel-resistant cancer cells is associated with feedback control of TUBB3 via FOXO3a-mediated ABCB1 regulation. Oncotarget |