Ian P. Street
Affiliations: | Systems Biology and Personalised Medicine | The Walter and Eliza Hall Institute of Medical Research, Parkville, Victoria, Australia |
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Sign in to add traineeAnderly C. Chueh | research scientist | 2016- | Walter and Eliza Hall Institute of Medical Research |
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Publications
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Sharma S, Chung CY, Uryu S, et al. (2023) Discovery of a highly potent, selective, orally bioavailable inhibitor of KAT6A/B histone acetyltransferases with efficacy against KAT6A-high ER+ breast cancer. Cell Chemical Biology |
Grohmann C, Walker F, Devlin M, et al. (2021) Preclinical small molecule WEHI-7326 overcomes drug resistance and elicits response in patient-derived xenograft models of human treatment-refractory tumors. Cell Death & Disease. 12: 268 |
Priebbenow DL, Leaver D, Nguyen N, et al. (2020) Discovery of Acylsulfonohydrazide-Derived Lysine Acetyltransferase (KAT6A) Inhibitors as Potent Senescence-Inducing Anti-Cancer Agents. Journal of Medicinal Chemistry |
MacPherson L, Anokye J, Yeung MM, et al. (2019) HBO1 is required for the maintenance of leukaemia stem cells. Nature |
AbuHammad S, Cullinane C, Martin C, et al. (2019) Regulation of PRMT5-MDM4 axis is critical in the response to CDK4/6 inhibitors in melanoma. Proceedings of the National Academy of Sciences of the United States of America |
Leaver D, Cleary B, Nguyen NT, et al. (2019) Discovery of benzoylsulfonohydrazides as potent inhibitors of the histone acetyltransferase KAT6A. Journal of Medicinal Chemistry |
Baell JB, Leaver DJ, Hermans SJ, et al. (2018) Inhibitors of histone acetyltransferases KAT6A/B induce senescence and arrest tumour growth. Nature |
Sonderegger S, Cerruti L, Tremblay C, et al. (2018) Small-Molecule Inhibition of PRMT5 Induces Translational Stress and p53 in JAK2V617F Mutant Myeloproliferative Neoplasms Blood. 132: 53-53 |
Toulmin E, Sonderegger S, Cerruti L, et al. (2018) PRMT5 Inhibition Selectively Targets Acute Myeloid Leukemia Stem Cells Though a p53-Dependent Mechanism Blood. 132: 4061-4061 |
Cursons J, Leuchowius KJ, Waltham M, et al. (2015) Stimulus-dependent differences in signalling regulate epithelial-mesenchymal plasticity and change the effects of drugs in breast cancer cell lines. Cell Communication and Signaling : Ccs. 13: 26 |