Eli E. Sercarz

Microbiology and Immunology University of California, Los Angeles, Los Angeles, CA 
"Eli Sercarz"
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Albert Coons grad student 1960 Harvard Medical School (ID Tree)
 (States of immunologic unresponsiveness in the mouse)


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Scheherazade Sadegh-Nasseri grad student UCLA
Suzanne Eaton grad student 1988 UCLA (FlyTree)
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Vong AM, Daneshjou N, Norori PY, et al. (2011) Spectratyping analysis of the islet-reactive T cell repertoire in diabetic NOD Igμ(null) mice after polyclonal B cell reconstitution. Journal of Translational Medicine. 9: 101
Rasche SS, Phillips M, McInerney MF, et al. (2011) IL-13Rα1 expression on β-cell-specific T cells in NOD mice. Diabetes. 60: 1716-25
Gabaglia CR, DeLaney A, Gee J, et al. (2010) Treatment combining RU486 and Ad5IL-12 vector attenuates the growth of experimentally formed prostate tumors and induces changes in the sentinel lymph nodes of mice Journal of Translational Medicine. 8
Maverakis E, Menezes JS, Ametani A, et al. (2010) Molecular mimics can induce a nonautoaggressive repertoire that preempts induction of autoimmunity. Proceedings of the National Academy of Sciences of the United States of America. 107: 2550-5
Dai YD, Sercarz EE. (2009) Antigen processing patterns determine GAD65-specific regulation vs. pathogenesis. Frontiers in Bioscience (Landmark Edition). 14: 344-51
Jensen KD, Sercarz EE, Gabaglia CR. (2009) Altered peptide ligands can modify the Th2 T cell response to the immunodominant 161-175 peptide of LACK (Leishmania homolog for the receptor of activated C kinase). Molecular Immunology. 46: 366-74
Dai YD, Marrero IG, Gros P, et al. (2009) Slc11a1 enhances the autoimmune diabetogenic T-cell response by altering processing and presentation of pancreatic islet antigens. Diabetes. 58: 156-64
Dai Y, Marrero I, Li Y, et al. (2009) OR.89. C-terminus of GAD65 is Capable of Inducing Diabetic Effector T Cells After Processing and Presentation of Apoptotic Islet Beta Cells in NOD Mice Clinical Immunology. 131: S37
Penitente R, Nicolò C, Van den Elzen P, et al. (2008) Administration of PLP139-151 primes T cells distinct from those spontaneously responsive in vitro to this antigen. Journal of Immunology (Baltimore, Md. : 1950). 180: 6611-22
Dai YD, Jensen KP, Marrero I, et al. (2008) N-terminal flanking residues of a diabetes-associated GAD65 determinant are necessary for activation of antigen-specific T cells in diabetes-resistant mice. European Journal of Immunology. 38: 968-76
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