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Geoffrey M. Cooper

Affiliations:

Biology

Boston University, Boston, MA, United States

Area:

Cancer Research

Website:

http://www.bu.edu/biology/people/faculty/cooper/

Google:

"Geoffrey M. Cooper"

Bio:

https://www.bu.edu/biology/people/faculty/faculty%20cvs/Cooper%20CV.pdf

Mean distance: 10.07

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Parents

Sheldon Greer	grad student	1973	University of Miami
(The effect of catabolism of halogenated pyrimidines on their utilization for nucleic acid synthesis)
Howard M. Temin	post-doc	1973-1975	UW Madison

Children

Alexandra Z. Adams	grad student	2006	Boston University
Jie Chen	grad student	2007	Boston University
Michael E. Schaffer	grad student	2007	Boston University
Julie R. Graham	grad student	2010	Boston University
Jolyon Terragni	grad student	2011	Boston University
Steven Mullenbrock	grad student	2012	Boston University
Channing J. Der	post-doc	1982	Harvard Medical School (Cell Biology Tree)

Collaborators

BETA: Related publications

Publications

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Adams KW, Kletsov S, Lamm RJ, et al. (2017) Role for Egr1 in the Transcriptional Program Associated with Neuronal Differentiation of PC12 Cells. Plos One. 12: e0170076
Nayak G, Cooper GM. (2012) p53 is a major component of the transcriptional and apoptotic program regulated by PI 3-kinase/Akt/GSK3 signaling. Cell Death & Disease. 3: e400
Mullenbrock S, Shah J, Cooper GM. (2011) Global expression analysis identified a preferentially nerve growth factor-induced transcriptional program regulated by sustained mitogen-activated protein kinase/extracellular signal-regulated kinase (ERK) and AP-1 protein activation during PC12 cell differentiation. The Journal of Biological Chemistry. 286: 45131-45
Tullai JW, Graham JR, Cooper GM. (2011) A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells. Cell Cycle (Georgetown, Tex.). 10: 3072-7
Terragni J, Nayak G, Banerjee S, et al. (2011) The E-box binding factors Max/Mnt, MITF, and USF1 act coordinately with FoxO to regulate expression of proapoptotic and cell cycle control genes by phosphatidylinositol 3-kinase/Akt/glycogen synthase kinase 3 signaling. The Journal of Biological Chemistry. 286: 36215-27
Tullai JW, Tacheva S, Owens LJ, et al. (2011) AP-1 is a component of the transcriptional network regulated by GSK-3 in quiescent cells. Plos One. 6: e20150
Graham JR, Hendershott MC, Terragni J, et al. (2010) mRNA degradation plays a significant role in the program of gene expression regulated by phosphatidylinositol 3-kinase signaling. Molecular and Cellular Biology. 30: 5295-305
Saxena UH, Owens L, Graham JR, et al. (2010) Prolyl isomerase Pin1 regulates transcription factor LSF (TFCP2) by facilitating dephosphorylation at two serine-proline motifs. The Journal of Biological Chemistry. 285: 31139-47
Graham JR, Tullai JW, Cooper GM. (2010) GSK-3 represses growth factor-inducible genes by inhibiting NF-κB in quiescent cells Journal of Biological Chemistry. 285: 4472-4480
Saxena UH, Powell CM, Fecko JK, et al. (2009) Phosphorylation by cyclin C/cyclin-dependent kinase 2 following mitogenic stimulation of murine fibroblasts inhibits transcriptional activity of LSF during G1 progression. Molecular and Cellular Biology. 29: 2335-45

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