James Jackson
Affiliations: | Biochemistry and Molecular Biology | Tulane School of Medicine |
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Lee H, Jung JH, Ko HM, et al. (2023) RNA-binding motif protein 10 inactivates c-Myc by partnering with ribosomal proteins uL18 and uL5. Proceedings of the National Academy of Sciences of the United States of America. 120: e2308292120 |
Chiu FY, Kvadas RM, Mheidly Z, et al. (2023) Could senescence phenotypes strike the balance to promote tumor dormancy? Cancer Metastasis Reviews |
Shahbandi A, Chiu FY, Ungerleider NA, et al. (2022) Breast cancer cells survive chemotherapy by activating targetable immune-modulatory programs characterized by PD-L1 or CD80. Nature Cancer |
Frey WD, Anderson AY, Lee H, et al. (2022) Phosphoinositide species and filamentous actin formation mediate engulfment by senescent tumor cells. Plos Biology. 20: e3001858 |
Park VS, Sun MJS, Frey WD, et al. (2022) Mouse model and human patient data reveal critical roles for Pten and p53 in suppressing POLE mutant tumor development. Nar Cancer. 4: zcac004 |
Shahbandi A, Rao SG, Anderson AY, et al. (2020) BH3 mimetics selectively eliminate chemotherapy-induced senescent cells and improve response in TP53 wild-type breast cancer. Cell Death and Differentiation |
Tonnessen-Murray CA, Jackson JG. (2020) Engulfment and cannibalism drive persistence of chemotherapy-treated tumor cells: can they be targeted? Molecular & Cellular Oncology. 7: 1688601 |
Tonnessen-Murray CA, Frey WD, Rao SG, et al. (2019) Chemotherapy-induced senescent cancer cells engulf other cells to enhance their survival. The Journal of Cell Biology |
Ungerleider NA, Rao SG, Shahbandi A, et al. (2018) Breast cancer survival predicted by TP53 mutation status differs markedly depending on treatment. Breast Cancer Research : Bcr. 20: 115 |
Tonnessen-Murray C, Ungerleider NA, Rao SG, et al. (2018) p53 Mediates Vast Gene Expression Changes That Contribute to Poor Chemotherapeutic Response in a Mouse Model of Breast Cancer. Translational Oncology. 11: 930-940 |